Toxoplasmic Retinochoroiditis
Mostrando 1-12 de 13 artigos, teses e dissertações.
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1. Serum levels of neurotrophic factors in active toxoplasmic retinochoroiditis
Abstract Toxoplasmic retinochoroiditis (TR) is the most common identifiable cause of posterior uveitis in Brazil. Response to treatment and clinical presentation may vary significantly. We assessed serum levels of brain-derived neurotrophic factor (BDNF), glial cell line-derived neurotrophic factor (GDNF), nerve growth factor (NGF), neurotrophin (NT)-3, and
Braz J Infect Dis. Publicado em: 2017-03
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2. Increased serum levels of soluble tumor necrosis factor receptor-2 (sTNFR2) in patients with active toxoplasmic retinochoroiditis
This study aimed to investigate the serum levels of the cytokine TNF-α and its soluble receptors (sTNFR1 and sTNFR2) in patients with toxoplasmosis retinochoroidits (TR) and controls. 37 patients with TR and 30 subjects with positive serology for toxoplasmosis but without history and signs of uveitis were included in this study. Serum concentrations of TNF-
Braz J Infect Dis. Publicado em: 2012-12
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3. Risk factors for Toxoplasma gondii infection and development of toxoplasmic retinochoroiditis. / Fatores de risco para infecção por Toxoplasma gondii e desenvolvimento da retinocoroidite toxoplásmica.
Toxoplasma gondii (T. gondii) infecta os seres humanos dentre outras vias, pelo trato gastrintestinal, local de expressão dos antígenos ABO por meio de interações epistáticas entre os genes ABO, Secretor e Lewis. A retinocoroidite toxoplásmica (RT), doença resultante desta infecção, é considerada a principal causa de uveíte posterior. Objetivo: Av
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 16/09/2011
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4. Candidate gene analysis of ocular toxoplasmosis in Brazil: evidence for a role for toll-like receptor 9 (TLR9)
Toxoplasma gondii infection is an important mediator of ocular disease in Brazil more frequently than reported from elsewhere. Infection and pathology are characterized by a strong proinflammatory response which in mice is triggered by interaction of the parasite with the toll-like receptor (TLR)/MyD88 pathway. A powerful way to identify the role of TLRs in
Memórias do Instituto Oswaldo Cruz. Publicado em: 2009-12
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5. Ocular toxoplasmosis: the influence of patient age
The influence of patient age on various features of ocular toxoplasmosis has been a subject of study for many years. The age at which Toxoplasma gondii infection occurs in different populations is related to socioeconomic factors and studies suggest that ocular toxoplasmosis is a more severe disease at the extremes of age. The prevalence of ocular involvemen
Memórias do Instituto Oswaldo Cruz. Publicado em: 2009-03
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6. Treating ocular toxoplasmosis: current evidence
The current treatment of ocular toxoplasmosis is controversial. The mainstay of treatment has been pyrimethamine and sulphonamides with or without systemic corticosteroids, but the actual evidence that antibiotics have a beneficial effect in recurrent toxoplasmic retinochoroiditis is unsupported by randomised placebo controlled trials. Thus far there have on
Memórias do Instituto Oswaldo Cruz. Publicado em: 2009-03
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7. DinÃmica da IgA secretora especÃfica na lÃgrima empacientes portadores de uveÃte posterior ativa presumivelmente por Toxoplasma gondii durante o primeiro trimestre da doenÃa
Toxoplasma gondii (T.gondii) is the main cause of posterior uveitis and toxoplasmic retinochoroiditis is considered the most common infection of the retina. Serological tests are important tools for the diagnosis of toxoplasmosis, but in ocular toxoplasmosis, usually there is no correlation between levels of serum antibodies and eye symptoms in the patient.
Publicado em: 2009
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8. Evidências sorológicas e experimentais da resposta autoimune humoral contra a retina em uveites causada por Toxoplasma gondii / Experimental and serological evidence of humoral autoimmune response against retina in Toxoplasma gondii uveitis
Ocular toxoplasmosis is attributed to the parasite, but autoimmunity could have a role in this process. Human sera, positive of anti-T. gondii IgG, show high levels of anti-retina IgG, measured by several antigens, as compared to T. gondii seronegative samples. Sera from patients with uveitis from other origins also had higher anti-retina abs levels. Challen
Publicado em: 2008
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9. Avaliação do perfil de quimiocinas e sua correlação com a intensidade do processo inflamatório nas retinocoroidites supostamente toxoplásmicas
Introduction: chemokines are a specific group of cytokines that has an important role in the angiogenesis and in leucocyte migration into the eye during intraocular inflammation. Purpose: to evaluate serum levels of the chemokines CCL2, CCL11, CXCL8, CXCL9 and CXCL10 in patients with presumed acute toxoplasmic retinochoroiditis, compare them to the patients
Publicado em: 2006
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10. Estudo morfológico das alterações vitreorretinianas em pacientes com retinocoroidite ativa supostamente causada pelo Toxoplasma Gondii: estudo retrospectivo pela Tomografia de Coerência Óptica (OCT 3).
Orefice, JL. Morphological study of vitreoretinal alterations in patients with active retinochoroiditis possibly caused by the Toxoplasma gondii . Prospective study by the optical coherence tomography (OCT 3) 2006. Thesis (Doctorate in Ophthalmology) - Medical School, Federal University of Minas Gerais, Belo Horizonte. Introduction: The ocular toxoplasmosis
Publicado em: 2006
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11. Utility of Tissue Culture for Detection of Toxoplasma gondii in Vitreous Humor of Patients Diagnosed with Toxoplasmic Retinochoroiditis
Laboratory recovery and confirmation of the etiologic agent in necrotizing retinochoroiditis are problematic. Tissue culture and intraocular antibody titers were compared as adjuncts to clinical diagnosis for toxoplasmic retinochoroiditis: the correlations were 91 and 67%, respectively. Isolation of Toxoplasma gondii may establish a definitive diagnosis in p
American Society for Microbiology.
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12. Treatment of active toxoplasmic retino-choroiditis.