Postprandial Lipemia
Mostrando 1-12 de 21 artigos, teses e dissertações.
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1. Body composition, but not insulin resistance, influences postprandial lipemia in patients with Turner's syndrome
ABSTRACT Objective: The aim of the present study was to examine the influence of body composition and insulin resistance on the magnitude of postprandial lipemia in patients with Turner's syndrome receiving oral versus transdermal estrogen replacement. Subjects and methods: Twenty-five patients with Turner's syndrome receiving oral or transdermal estrogen
Arch. Endocrinol. Metab.. Publicado em: 2020-12
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2. Plasma Renin in Women Using and not Using Combined Oral Contraceptive
Abstract Backgroud: Recent studies show that women on combined oral contraceptives (COC) present abnormal fasting lipid profile, increased postprandial lipemia, plasma C-reactive protein (CRP) and blood pressure (BP) compared to women not on combined oral contraceptives. Plasma renin is one of the factors responsible for abnormal BP. Objectives: To assess
Int. J. Cardiovasc. Sci.. Publicado em: 2020-05
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3. Covid-19 and Safety in the Cath Lab: Where We Are and Where We Are Headed
Abstract Backgroud: Recent studies show that women on combined oral contraceptives (COC) present abnormal fasting lipid profile, increased postprandial lipemia, plasma C-reactive protein (CRP) and blood pressure (BP) compared to women not on combined oral contraceptives. Plasma renin is one of the factors responsible for abnormal BP. Objectives: To assess
Int. J. Cardiovasc. Sci.. Publicado em: 2020-05
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4. Orange juice with a high-fat meal prolongs postprandial lipemia in apparently healthy overweight/obese women
ABSTRACT Objective We investigated the postprandial response of lipid markers to a high-fat meal (HFM) with two different beverages in apparently healthy normal-weight and overweight/obese women. Subjects and methods This crossover, randomized study enrolled 36 women, of whom 21 had normal weight (body mass index [BMI] 22 ± 1.8 kg/m2) and 15 had overwei
Arch. Endocrinol. Metab.. Publicado em: 24/11/2016
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5. Ingestão de ácidos graxos monoinsaturados e metabolismo lipídico
Resumo Esta revisão teve como objetivo apresentar e discutir os achados mais recentes do efeito dos ácidos graxos monoinsaturados (AGMI) sobre marcadores plasmáticos do metabolismo lipídico em estudos pós-prandiais e de intervenção clínica nutricional. Realizou-se busca em diferentes bases de dados entre 2010 e 2014, usando os seguintes termos de ind
J. vasc. bras.. Publicado em: 2016-03
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6. The influence of intense intermittent versus moderate continuous exercise on postprandial lipemia
INTRODUCTION: Postprandial lipemia is characterized by an increased concentration of circulating lipids after fat intake and is an independent risk factor for cardiovascular disease. Exercise is known to reduce postprandial lipemia and its negative clinical outcomes. OBJECTIVE: This study investigated the effect of intense intermittent versus moderate contin
Clinics. Publicado em: 2011
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7. Responses of postprandial lipemia in young men, after continuous and intermittent exercise / Respostas da lipemia pós-prandial em homens jovens, após exercício contínuo e intermitente
Background: Increased plasma triglyceride-rich lipoprotein (chylomicrons, VLDL and LDL) concentration, as well as the delay in their removal, has been associated with coronary artery diseases (CVD). The postprandial lipaemia is related to peripheral vascular changes, stimulated by inflammatory markers and enhanced by oxidative metabolism of low density lipop
Publicado em: 2010
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8. Associação do Genótipo da apolipoproteína E e diferentes intensidade de exercício na lipemia pós-prandial de homens jovens
Introduction: Postprandial lipemia (PPL) refers to a series of metabolic events related to the increased concentration of circulating lipids, which occurs after the consumption of fats intake. It is an indirect stimulation of atherosclerosis, since it promotes the formation of atherosclerotic plaques through the wall of the arteries. It is associated with in
Publicado em: 2010
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9. Lipemia postrandial in the alterations of the activities in the cholesterol ester transfer protein or hepatic lipase in human and rodents / Lipemia pos-prandial nas alterações das atividades da proteina de tranferencia de esteres de colesterol ou da lipase hepatica em humanos e em roedores
The postprandial response to a fat overload presents proatherogenic direct effects with remanescent lipoproteins deposition in arterial walls and indirect on the triglycerides-rich lipoprotein metabolism. Alterations during the postprandial lipemia are related with the activities of cholesteryl ester transfer protein and hepatic lipase. These two proteins po
Publicado em: 2007
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10. Lipideos sericos no periodo pos-alimentar : padrões da resposta lipemica e sua modulação pelos sexos
Atherosclerosis is a multifactorial, immuno-inflammatory and progressive disease of the arterial wall. Postalimentary lipemia is associated with an increased risk of coronary artery disease. The metabolic and pro-oxidant effects in fasting and postprandial (2, 4, 6 and 8h) periods were measured in an adult population ofboth sexes, without risk factors for co
Publicado em: 2004
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11. High density lipoprotein2. Relationship of the plasma levels of this lipoprotein species to its composition, to the magnitude of postprandial lipemia, and to the activities of lipoprotein lipase and hepatic lipase.
Lipoprotein lipase (LPL) activity in postheparin plasma of 38 normolipidemic volunteers was related to the magnitude of postprandial lipemia after a fat meal, to triglyceride content of high density lipoprotein2 (HDL2), to hepatic lipase (HL) activity, and to HDL2 levels. LPL activity correlated indirectly with lipemia, triglyceride content of HDL2, HL activ
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12. Heterozygous lipoprotein lipase deficiency due to a missense mutation as the cause of impaired triglyceride tolerance with multiple lipoprotein abnormalities.
In 16 members of two Austrian families affected by a missense mutation at codon 188 of the lipoprotein lipase (LPL) gene (8 heterozygous and 8 normal subjects), carrier status for the mutation as determined by DNA analysis was related to LPL activity in postheparin plasma, to the magnitude of postprandial lipemia, and to concentration, composition, and size