Non Alcoholic Fatty Liver Disease Nafld
Mostrando 13-24 de 25 artigos, teses e dissertações.
-
13. Can FIB4 and NAFLD fibrosis scores help endocrinologists refer patients with non-alcoholic fat liver disease to a hepatologist?
ABSTRACT Objective The objective of this study is to evaluate the performance of mathematical models used in non-invasive diagnosis of liver fibrosis in nonalcoholic fatty liver disease (NAFLD) patients to determine when the patient needs to be referred to a hepatologist. Subjects and methods Patients referred by endocrinologists to the liver outpatient
Arch. Endocrinol. Metab.. Publicado em: 13/02/2017
-
14. Effect of curcumin on visfatin and zinc-α2-glycoprotein in a rat model of non-alcoholic fatty liver disease
ABSTRACT PURPOSE: To investigate the effect of curcumin on visfatin and zinc-α2-glycoprotein (ZAG) expression levels in rats with non-alcoholic fatty liver disease (NAFLD). METHODS: Fifty-six male rats were randomly divided into a control group (n=16) and model group (n=40) and were fed on a normal diet or a high-fat diet, respectively. Equal volumes of
Acta Cir. Bras.. Publicado em: 2016-11
-
15. FT3/FT4 ratio predicts non-alcoholic fatty liver disease independent of metabolic parameters in patients with euthyroidism and hypothyroidism
OBJECTIVE: This study was performed to evaluate the effects of metabolic parameters and thyroid dysfunction on the development of non-alcoholic fatty liver disease (NAFLD). METHODS: The current study evaluated a total of 115 patients, 75 female and 40 male. Physical examination and anthropometric measurements were applied to all participants. Hypothyroidis
Clinics. Publicado em: 2016-04
-
16. The spectrum of non alcoholic fatty liver disease in morbidly obese patients: prevalence and associate risk factors
PURPOSE: To determine the prevalence of non alcoholic fatty liver disease (NAFLD) and non alcoholic steatohepatitis (NASH) in morbidly obese patients undergoing bariatric surgery and to identify risk factors associated with the disease spectrum. METHODS: Liver biopsy was performed in 60 patients who underwent bariatric surgery, after other causes of liver di
Acta Cir. Bras.. Publicado em: 2013-11
-
17. Effect and the probable mechanisms of silibinin in regulating insulin resistance in the liver of rats with non-alcoholic fatty liver
Our previous study has shown that reduced insulin resistance (IR) was one of the possible mechanisms for the therapeutic effect of silibinin on non-alcoholic fatty liver disease (NAFLD) in rats. In the present study, we investigated the pathways of silibinin in regulating hepatic glucose production and IR amelioration. Forty-five 4- to 6-week-old male Spragu
Braz J Med Biol Res. Publicado em: 15/03/2013
-
18. Tratamento interdisciplinar em adolescentes obesos com esteatose hepatica não alcoolica: papel dos neuropeptideos e adipocinas pro e anti-inflamatorias / Interdisciplinary treatment in obese adolescents with NAFLD: role of neuropeptides and adipokines pro and anti-inflammatory
Introdução: A complexidade da fisiopatologia na Esteatose Hepática Não Alcóolica (EHNA) envolve uma interface entre as adipocinas e a regulação neuroendócrina do balanço energético, incluindo o papel do sistema Neuropeptídeo Y/Proteína agouti relacionada. Objetivo: estabelecer a relação entre a razão NeuropeptídeoY/Proteína relacionada Agout
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 31/08/2011
-
19. Effects of oral administration of S-nitrosothiols and knectic evaluation of the kinetic propensity and resistance to diet induced obessity / Efeitos da administração orogástrica de s-nitrosotióis e avaliação cinética da propensão e resistência à obsidade induzida por dieta
In the present study, the nitric oxide (NO)-associated physiological actions obtained topically on the gastric mucosa and systemically in oral administrations of two NO donors-S-nitrosothiols (RSNOs), S-nitrosoglutathione (GSNO) and S-nitroso-N-acetylcysteine (SNAC) were investigated in animal models. The topical ex vivo administration of RSNOs solutions on
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 15/07/2011
-
20. FreqÃÃncia e fatores de risco da DoenÃa HepÃtica Gordurosa NÃo AlcoÃlica em pacientes com Diabetes Mellitus tipo 2 atendidos no HC-UFPE
Proposal: In the Western world, where the lifestyle has contributed to an increase in obesity, dyslipidemia and diabetes, Non-Alcoholic Fatty Liver Disease (NAFLD) has already become the most common chronic liver disease, despite studies about it having only begun in the 1980s. NAFLD is closely linked to cases of insulin resistance (IR), and when linked with
Publicado em: 2008
-
21. Aspectos da sÃndrome metabÃlica na doenÃa hepÃtica gordurosa nÃo-alcoÃlica em pacientes obesos mÃrbidos
Obesity has become an epidemic disease in whose prevalence in going up throughout the world, particularly in its malignant form, the severe obesity. The severe obesity represents the Insulin Resistance Syndrome and its high risks of morbi-mortality to it related like diabetes, cancers, miocardial infartation and liver damage: liver steatosis, non alcoholic s
Publicado em: 2003
-
22. A growing burden: the pathogenesis, investigation and management of non‐alcoholic fatty liver disease
Non‐alcoholic fatty liver disease (NAFLD) is the most common hepatic disorder in western countries, and its incidence is increasing. This review outlines the significant health burden posed by NAFLD and discusses what is presently known about its pathogenesis, including the roles of the metabolic syndrome, obesity, insulin resistance, hepatic steatosis, re
BMJ Group.
-
23. Transient elastography in patients with non‐alcoholic fatty liver disease (NAFLD)
BMJ Group.
-
24. High Fat Diet Induced Hepatic Steatosis Establishes a Permissive Microenvironment for Colorectal Metastases and Promotes Primary Dysplasia in a Murine Model
Non-alcoholic fatty liver disease (NAFLD), which includes steatosis and its progression to non-alcoholic steatohepatitis, is a liver disorder of increasing clinical significance. Here we characterize a murine model of high fat diet-induced NAFLD with progression from liver steatosis to histological features compatible with steatohepatitis and more advanced s
American Society for Investigative Pathology.