Leukoencephalopathy Progressive Multifocal
Mostrando 1-12 de 80 artigos, teses e dissertações.
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1. Progressive multifocal leukoencephalopathy in a patient with relapsed chronic lymphocytic leukemia treated with Ibrutinib
Hematology, Transfusion and Cell Therapy. Publicado em: 2022
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2. Possible patient to patient transmission of progressive multifocal leukoencephalopathy among kidney-transplant patients
RESUMO Este estudo teve como objetivo caracterizar e estimar a divergência genética entre doze espécimes do gênero Sansevieria da Coleção da Universidade Federal do Piauí (UFPI). Utilizou-se o delineamento experimental inteiramente ao acaso, com três repetições, sendo a parcela constituída por quatro plantas. Para caracterização morfológica for
Braz J Infect Dis. Publicado em: 2020-10
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3. JC virus/human immunodeficiency virus 1 co-infection in the Brazilian Amazonian region
Abstract JC virus (JCV) is a member of the Polyomaviridae family and is associated to a severe disease known as progressive multifocal leukoencephalopathy, PML, which is gradually increasing in incidence as an opportunistic infection among AIDS patients. The present study aimed to investigate the occurrence of JCV among HIV-1 carriers including their types a
Braz J Infect Dis. Publicado em: 2016-08
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4. JC virus-associated central nervous system diseases in HIV-infected patients in Brazil: clinical presentations, associated factors with mortality and outcome
INTRODUCTION: Several presentations of neurologic complications caused by JC virus (JCV) in human immunodeficiency virus (HIV)-infected patients have been described and need to be distinguished from the "classic" form of progressive multifocal leukoencephalopathy (PMl). The objectives of this study were: 1) to describe the spectrum and frequency of presentat
Brazilian Journal of Infectious Diseases. Publicado em: 2012-04
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5. Progressive multifocal leukoencephalopathy restricted to the posterior fossa in a patient with systemic lupus erythematosus
Progressive multifocal leukoencephalopathy is a neurological infectious disease caused by the John Cunningham polyomavirus (JCV), an opportunistic agent with worldwide distribution. This disease is frequently seen in immunosuppresed patients and rarely associated with systemic lupus erythematosus. In the central nervous system PML demyelinating lesions occur
Brazilian Journal of Infectious Diseases. Publicado em: 2011-12
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6. Human polyomaviruses JC and BK in the urine of Brazilian children and adolescents vertically infected by HIV
The aim of this study was to characterize the urinary excretion of the BK (BKV) and JC (JCV) human polyomaviruses in a cohort of human immunodeficiency virus (HIV)-infected children and adolescents. One hundred and fifty-six patients were enrolled: Group I included 116 HIV-infected children and adolescents [median age = 11.4 years (y); range 1-22 y]; Group I
Memórias do Instituto Oswaldo Cruz. Publicado em: 2011-12
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7. Polyoma BK virus: an emerging opportunistic infectious agent of the human central nervous system
BK virus, a double-stranded DNA virus, is a member of the Polyomaviridae family which is known to infect humans. Clinical evidence of disease is mostly encountered in immunosuppressed individuals such as AIDS patients or those who undergo renal or bone marrow transplantation where complications associated with BKV infection manifest commonly as a polyomaviru
Brazilian Journal of Infectious Diseases. Publicado em: 2011-06
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8. Genotype distribution of human polyomavirus JC in AIDS patients with and without progressive multifocal leukoencephalopathy, in Sao Paulo, Brazil / Distribuição genotípica do poliomavirus humano JC em pacientes com aids, com e sem leucoencefalopatia multifocal progressiva, em São Paulo, Brasil
A relação entre os diferentes genótipos do VJC e a patogênese da LEMP permanece ainda uma questão controversa. O presente estudo visou a caracterização genotipica do vírus JC (VJC) e sua associação com a leucoencefalopatia multifocal progressiva (LEMP) em pacientes com aids em São Paulo, no período de 2000 a 2008. Foram avaliadas 51 amostras de l
Publicado em: 2010
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9. Detecção do DNA do Poliomavírus Humano JC em amostras de líquido cefalorraquidiano de pacientes com AIDS e lesões não expansivas de substância branca do sistema nervoso central / Detection of human polyomavirus JC in cerebrospinal fluid samples from aids patients with non-expansive focal lesions of CNS white matter
Focal neurological diseases in aids patients can be caused by a range of opportunistic pathogens such as Toxoplasma gondii, EBV-associated primary CNS lymphomas, viral encephalitis (CMV, HSV, VZV) and JC virus causing the progressive multifocal leukoencephalopathy (PML). In the present study, we evaluated the detection of JC virus DNA in CSF samples from aid
Publicado em: 2004
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10. Diagnostic value of detecting JC virus DNA in cerebrospinal fluid of patients with progressive multifocal leukoencephalopathy.
JC virus DNA was detected by PCR in the cerebrospinal fluid of 17 of 23 (73.9%) patients with confirmed cases of progressive multifocal leukoencephalopathy and 2 of 48 (4.2%) controls without progressive multifocal leukoencephalopathy. The sensitivity and specificity of this PCR were 74 and 95.8%, respectively, while the positive and negative predictive valu
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11. Cortical and subcortical JC virus infection: two unusual cases of AIDS associated progressive multifocal leukoencephalopathy.
Two patients with AIDS and progressive neurological syndromes had necropsies that identified JC virus infection of the cerebral or cerebellar cortex. The unusual presentation of progressive multifocal leukoencephalopathy with grey matter involvement and normal cerebral imaging is discussed.
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12. Detection of JC Virus in Cerebrospinal Fluid (CSF) Samples from Patients with Progressive Multifocal Leukoencephalopathy but Not in CSF Samples from Patients with Herpes Simplex Encephalitis, Enteroviral Meningitis, or Multiple Sclerosis
JC virus (JCV) DNA was detected in cerebrospinal fluid (CSF) samples from patients with progressive multifocal leukoencephalopathy (PML) but not in CSF samples from patients with herpes simplex encephalitis, enteroviral meningitis, or multiple sclerosis. This suggests that inflammatory processes in the brain do not necessarily reactivate JCV, which further s
American Society for Microbiology.