Voltage Regulators
Mostrando 13-24 de 32 artigos, teses e dissertações.
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13. Methodology for calculation of technical losses in each segment of the distibution system. / Metodologia para cálculo de perdas técnicas por segmento do sistema de distribuição.
This work focuses on the development of a new methodology for calculating technical losses within the different segments of the electric distribution system. In order to evaluate technical losses the electrical system was divided into eight segments: energy meter, customer connections, secondary feeder, distribution transformer, primary feeder, distribution
Publicado em: 2001
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14. AGREGAÇÃO DINÂMICA DE MODELOS DE ESTABILIZADORES APLICADOS EM REGULADORES DE TENSÃO E CÁLCULO DE EQUIVALENTES DINÂMICOS / DYNAMIC AGGREGATION OF POWER SYSTEM STABILIZER MODELS APPLIED IN VOLTAGE REGULATORS AND CALCULATION OF DYNAMIC EQUIVALENTS
The calculation of coherency-based dynamic equivalents has three main steps: the identification of coherent generators, the network reduction and the dynamic aggregation of the coherent generating unit models. This dissertation deals with the problem of dynamic aggregation of power system stabilizer models to calculate coherency-based dynamic equivalents for
Publicado em: 1999
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15. PLANNING OF A DISTRIBUITION NETWORK / PLANEJAMENTO DE REDES DE DISTRIBUIÇÃO
This work presentes a methodology and a digital computer program for the expansion planning of a distribuition network that allows to determine, for exemple, the location of capacitors bank and voltage regulators and/or automatic-boosters, voltage gain and redution of electrical losses with the changing of conductors. The proposed methodology is based on the
Publicado em: 1992
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16. METHODS FOR THE ANALYSIS OF DINAMICALLY UNSTABLE MULTIMACHINE SYSTEMS / MÉTODOS PARA ANÁLISE DE SISTEMAS MULTIMÁQUINAS DINAMICAMENTE INSTÁVEIS
The practices normally used in classical digital programs for oscillation mode analysis in dynamically unstable multimachine systems, do not use, in such way a general methodology that allows identification, without a great deal of trial and error, wich machines, are more effectives in providing damping to the system through suplementary signal application i
Publicado em: 1976
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17. MODELLING OF VOLTAGE REGULATORS / REPRESENTAÇÃO DE REGULADORES DE TENSÃO
Neste trabalho é estudado um meio de se determinar os parâmetros de um regulador de tensão. È também discutida a determinação do modelo para o regulador. São mostrados exemplos diferentes aos reguladores de ação não contínua. Foi desenvolvido um programa digital para simular os testes de campo realizados com os reguladores.
Publicado em: 1975
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18. Molecular basis of fast inactivation in voltage and Ca2+-activated K+ channels: A transmembrane β-subunit homolog
Voltage-dependent and calcium-sensitive K+ (MaxiK) channels are key regulators of neuronal excitability, secretion, and vascular tone because of their ability to sense transmembrane voltage and intracellular Ca2+. In most tissues, their stimulation results in a noninactivating hyperpolarizing K+ current that reduces excitability. In addition to noninactivati
The National Academy of Sciences.
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19. Ethanol selectively blocks a noninactivating K+ current expressed in Xenopus oocytes.
There is presently a debate regarding the relative merits of lipid-based and protein-based theories of anesthesia and the action of ethanol in the central nervous system. Voltage-sensitive K+ channels play a key role as regulators of neuronal electrical activity and are potential targets of ethanol and other anesthetic agents. We investigated the action of l
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20. Antisense oligonucleotides against rat brain α1E DNA and its atrial homologue decrease T-type calcium current in atrial myocytes
Low voltage-activated, or T-type, calcium currents are important regulators of neuronal and muscle excitability, secretion, and possibly cell growth and differentiation. The gene (or genes) coding for the pore-forming subunit of low voltage-activated channel proteins has not been unequivocally identified. We have used reverse transcription–PCR to identify
The National Academy of Sciences of the USA.
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21. PIP3 inhibition of RGS protein and its reversal by Ca2+/calmodulin mediate voltage-dependent control of the G protein cycle in a cardiac K+ channel
Regulators of G protein signaling (RGS) accelerate intrinsic GTP hydrolysis on α subunits of trimeric G proteins and play crucial roles in the physiological regulation of G protein-mediated cell signaling. The control mechanisms of the action of RGS proteins per se are poorly clarified, however. We recently showed a physiological mode of action of a RGS pro
The National Academy of Sciences.
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22. Regulation and pharmacology of the mitochondrial permeability transition pore
The ‘mitochondrial permeability transition', characterized by a sudden induced change of the inner mitochondrial membrane permeability for water as well as for small substances (≤1.5 kDa), has been known for three decades. Research interest in the entity responsible for this phenomenon, the ‘mitochondrial permeability transition pore’ (mPTP), has dra
Oxford University Press.
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23. Direct interaction with a nuclear protein and regulation of gene silencing by a variant of the Ca2+-channel β4 subunit
The β subunits of voltage-gated Ca2+ channels are known to be regulators of the channels' gating properties. Here we report a striking additional function of a β subunit. Screening of chicken cochlear and brain cDNA libraries identified β4c, a short splice variant of the β4 subunit. Although β4c occurs together with the longer isoforms β4a or β4b in t
The National Academy of Sciences.
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24. RGS3 mediates a calcium-dependent termination of G protein signaling in sensory neurons
G proteins modulate synaptic transmission. Regulators of G protein signaling (RGS) proteins accelerate the intrinsic GTPase activity of Gα subunits, and thus terminate G protein activation. Whether RGS proteins themselves are under cellular control is not well defined, particularly in native cells. In dorsal root ganglion neurons overexpressing RGS3, we
National Academy of Sciences.