Purkinje Cells
Mostrando 1-12 de 321 artigos, teses e dissertações.
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1. Impressions of the chronic 900-MHz electromagnetic field in the prenatal period on Purkinje cells in male rat pup cerebella: is it worth mentioning?
SUMMARY OBJECTIVE: The aim of this study was to examine the changes on the Purkinje cells in the cerebella of male rat pups born to pregnant dams that were exposed to an electromagnetic field in the prenatal period. METHODS: The first stage of the study involved 12 Sprague-Dawley rats, 6 male and 6 female, weighing between 180 and 250 g. The female rats in
Revista da Associação Médica Brasileira. Publicado em: 2022
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2. Rabies encephalitis and extra-neural manifestations in a patient bitten by a domestic cat
ABSTRACT Rabies encephalitis is a fatal zoonotic viral disease transmitted to humans either by domestic animals like dogs and cats or by wild animals like bats, skunks and raccoons. We present the case of a 25-year-old woman admitted due to behavioral disorders, generalized paresthesiasand acute respiratory deterioration compatible with a respiratory distres
Rev. Inst. Med. trop. S. Paulo. Publicado em: 17/01/2020
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3. Neurological diseases in cattle caused by plants and mycotoxins in Santa Catarina state, Brazil
RESUMO: Descrevem-se os aspectos epidemiológicos e clinico-patológicos de 25 surtos de enfermidade neurológica em bovinos no estado de Santa Catarina causadas por plantas e micotoxinas. Destes, seis corresponderam a intoxicação por Sida carpinifolia, cinco por Solanum fastigiatum, cinco por Phalaris angusta, três por Claviceps paspali, três por Clavic
Pesq. Vet. Bras.. Publicado em: 02/05/2019
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4. Myosin Va is developmentally regulated and expressed in the human cerebellum from birth to old age
Myosin Va functions as a processive, actin-based motor molecule highly enriched in the nervous system, which transports and/or tethers organelles, vesicles, and mRNA and protein translation machinery. Mutation of myosin Va leads to Griscelli disease that is associated with severe neurological deficits and a short life span. Despite playing a critical role in
Braz J Med Biol Res. Publicado em: 2013-02
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5. Material particulado fino presente no ar da cidade de São Paulo promove alterações nas células de Purkinje: um estudo experimental em embrião de galinha / Urban fine particles induce alterations in Purkinje cells: an experimental study in chick embryo
Air pollutants are associated to several diseases including those related to central nervous system. The aim of this work was to evaluate the effect of urban fine particles (PM2.5) in chicken embryo Purkinje cells. A single dose of PM2.5 suspension in two different concentrations (1.5 or 20.0 Pg.100Pl-1) was injected in fresh laid fertilized eggs on E0. Cont
Publicado em: 2011
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6. Expressão dos receptores de glutamato do tipo NMDA em aves durante o desenvolvimento e envelhecimento / Expression of NMDA type glutamate receptors in the cerebellum of birds during aging
Aging is a physiologic process that takes place in a non-synchronic way in different areas of the brain and which can be defined as a dynamic and progressive process. There are morphological, functional and biochemical alterations which can lead to cognitive, affective, learning, memory and motor deficiencies. Since the cerebellum and the glutamatergic neuro
Publicado em: 2008
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7. Dinamica e controle não lineares de um sistema neuronal ideal e nã-ideal / No-linear dynamics and control of an ideal and non-ideal neural system
In this work it is studied the plasmatic membrane behavior through an electric circuit. The electric model was developed by Hodgkin and Huxley at 1952 and it treats of the variation of the amount of time related with the potassium and sodium conductance s in the squid axon, this model has served as an archetype for mannering mathematical model of eletrophysi
Publicado em: 2005
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8. Caracterização de um receptor para o prion atraves da teoria da hidropaticidade complementar dos amino acidos : evidencias da inteprion-laminina
Prions are slow infectious pathogens which cause transmissible neurodegenerative Jackob disease, diseases in both humans (kuru, Creutzfeldt-Gerstmann-Straussler-Scheinker syndrome and familial fatal insomnia) and animals (scrapie, bovine spongiform encephalopaty, transmissible mink encephalopaty and chronic wasting disease). The infectious prion protein (Prp
Publicado em: 1996
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9. Responses of Purkinje cells and mossy fibres in the flocculus of the monkey during sinusoidal movements of a visual pattern.
1. Discharges of Purkinje cells (P cells) and mossy fibres were recorded from the cerebellar flocculus of monkeys trained to fixate a stationary visual target. The units were tested with a sinusoidally moving random dot pattern (background) which was projected on an entire screen or on part of it. The receptive field organization of the units was tested by c
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10. Contribution of transplanted bone marrow cells to Purkinje neurons in human adult brains
We show here that cells within human adult bone marrow can contribute to cells in the adult human brain. Cerebellar tissues from female patients with hematologic malignancies, who had received chemotherapy, radiation, and a bone marrow transplant, were analyzed. Brain samples were obtained at autopsy from female patients who received male (sex-mismatched) or
The National Academy of Sciences.
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11. Mifepristone (RU486) protects Purkinje cells from cell death in organotypic slice cultures of postnatal rat and mouse cerebellum
Mifepristone (RU486), which binds with high affinity to both progesterone and glucocorticosteroid receptors (PR and GR), is well known for its use in the termination of unwanted pregnancy, but other activities including neuroprotection have been suggested. Cerebellar organotypic cultures from 3 to 7 postnatal day rat (P3—P7) were studied to examine the
National Academy of Sciences.
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12. Expression of truncated PrP targeted to Purkinje cells of PrP knockout mice causes Purkinje cell death and ataxia
PrP knockout mice with disruption of only the PrP-encoding region (Zürich I-type) remain healthy, whereas mice with deletions extending upstream of the PrP-encoding exon (Nagasaki-type) suffer Purkinje cell loss and ataxia, associated with ectopic expression of Doppel in brain, particularly in Purkinje cells. The phenotype is abrogated by co-expression of
Oxford University Press.