Liver Transplantation Pathology
Mostrando 1-10 de 10 artigos, teses e dissertações.
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1. Refractory ascites and graft dysfunction in early renal transplantation
Resumo A ocorrência de ascite no pós-Transplante Renal (TR) é infrequente, podendo ser consequência de complicações cirúrgicas ou médicas. Caso clínico: 61 anos, masculino, antecedentes de hipertensão arterial, carcinoma da língua e hábitos alcoólicos 12-20g/dia. Doença renal crônica secundária à doença renal poliquística autossômica domi
J. Bras. Nefrol.. Publicado em: 18/03/2019
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2. Escala Perme como preditor de funcionalidade e complicações após a alta da unidade de terapia intensiva em pacientes submetidos a transplante hepático
RESUMO Objetivo: Avaliar a pontuação da escala Perme de mobilidade como preditor de funcionalidade e complicações no pós-operatório de pacientes submetidos a transplante hepático. Métodos: A amostra foi composta por 30 pacientes que realizaram transplante hepático. Os pacientes foram avaliados em dois momentos, a fim de verificar a percepção da
Rev. bras. ter. intensiva. Publicado em: 2019-03
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3. Observation of the effect of bone marrow mesenchymal stem cell transplantation by different interventions on cirrhotic rats
Bone marrow mesenchymal stem cells (BMSCs) transplantation has attracted attention for the treatment of liver cirrhosis and end-stage liver diseases. Therefore, in this study, we evaluated the effect of different methods of BMSCs transplantation in the treatment of liver cirrhosis in rats. Seventy-two male Sprague-Dawley rats were divided into 7 groups: 10 w
Braz J Med Biol Res. Publicado em: 25/02/2019
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4. O desempenho da tomografia computadorizada na quantificação da esteatose hepática / The performance of computed tomography in quantifying hepatic steatosis
INTRODUCTION: Hepatic steatosis is a metabolic disease characterized by accumulated fat in hepatocytes. Among its causes, chronic alcoholism, obesity, type II diabetes mellitus and syndrome metabolic. The interest in detecting and quantifying hepatic steatosis has grown in the past years due to higher prevalence and advances in hepatic surgical technical. Qu
Publicado em: 2010
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5. "Hepatite colestática associada ao vírus da hepatite C pós-transplante hepático: estudo virológico, histopatológico e imuno-histoquímico" / Severe recurrent cholestatic hepatitis after liver transplantation : virological, histological and immuno-histochemical evaluation
Following liver transplantation (OLT) HCV-related disease severity is highly variable, with a minority of cases progressing to an extremely severe form of cholestatic hepatitis, in which the pathogenesis is not yet understood. We aim to compare virological, histological and immunohistological changes in patients developing mild and severe post-OLT HCV recurr
Publicado em: 2004
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6. Technique and results of biliary reconstruction using side-to-side choledochocholedochostomy in 300 orthotopic liver transplants.
OBJECTIVE: The authors evaluated the complication rate and outcome of side-to-side common bile duct anastomosis after human orthotopic liver transplantation. SUMMARY BACKGROUND DATA: Early and late biliary tract complications after orthotopic liver transplantation remain a serious problem, leading to increased morbidity and mortality. Commonly performed tech
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7. The lethal effects of transplantation of Socs1-/- bone marrow cells into irradiated adult syngeneic recipients
Injection of neonatal bone marrow cells from mice lacking the gene encoding suppressor of cytokine signaling 1 (SOCS1) into irradiated syngeneic 129/Sv or C57BL/6 mice led to a decreased survival, more rapidly occurring in 129/Sv than in C57BL/6 mice. Moribund mice did not exhibit the acute or chronic diseases developed by Socs1-/- mice but developed a
National Academy of Sciences.
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8. Bone marrow transplantation reproduces the tristetraprolin-deficiency syndrome in recombination activating gene-2 (-/-) mice. Evidence that monocyte/macrophage progenitors may be responsible for TNFalpha overproduction.
Tristetraprolin-deficient [TTP (-/-)] mice exhibit a complex syndrome of myeloid hyperplasia, cachexia, dermatitis, autoimmunity, and erosive arthritis. Virtually the entire syndrome can be prevented by the repeated injection of anti-TNFalpha antibodies (Taylor, G.A., E. Carballo, D.M. Lee, W.S. Lai, M.J. Thompson, D.D. Patel, D.I. Schenkman, G.S. Gilkeson,
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9. Reprogramming erythroid cells for lysosomal enzyme production leads to visceral and CNS cross-correction in mice with Hurler syndrome
Restricting transgene expression to maturing erythroid cells can reduce the risk for activating oncogenes in hematopoietic stem cells (HSCs) and their progeny, yet take advantage of their robust protein synthesis machinery for high-level protein production. This study sought to evaluate the feasibility and efficacy of reprogramming erythroid cells for produc
National Academy of Sciences.
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10. Differential effects of the absence of interferon-gamma and IL-4 in acute graft-versus-host disease after allogeneic bone marrow transplantation in mice.
Graft-versus-host disease (GVHD), in which immunocompetent donor cells attack the host, remains a major cause of morbidity after allogeneic bone marrow transplantation (BMT). To understand the role of cytokines in the pathobiology of GVHD, we used cytokine knockout (KO) mice as a source of donor T cells. Two different MHC-disparate strain combinations were e