Hemodynamics And Vascular Function
Mostrando 1-12 de 13 artigos, teses e dissertações.
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1. Efeito da Justicia acuminatissima, Sara Tudo do Amazonas, na injúria renal aguda isquêmica: estudo experimental
RESUMO Objetivo Avaliar o efeito da Justicia acuminatissima , Sara Tudo do Amazonas, na função renal, na hemodinâmica renal, no perfil oxidativo e na histologia renal em ratos com injúria renal aguda isquêmica. Método Ensaio pré-clínico com ratos Wistar, adultos, machos (250-350 g), distribuídos nos grupos Sham, Isquemia e Isquemia + Sara Tudo.
Rev. esc. enferm. USP. Publicado em: 19/08/2019
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2. Endothelin-1 receptor antagonists protect the kidney against the nephrotoxicity induced by cyclosporine-A in normotensive and hypertensive rats
Cyclosporin-A (CsA) is an immunosuppressant associated with acute kidney injury and chronic kidney disease. Nephrotoxicity associated with CsA involves the increase in afferent and efferent arteriole resistance, decreased renal blood flow (RBF) and glomerular filtration. The aim of this study was to evaluate the effect of Endothelin-1 (ET-1) receptor blockad
Braz J Med Biol Res. Publicado em: 11/12/2017
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3. Relação entre a capacidade vasodilatadora periférica e os mecanismos hemodinâmicos da hipotensão pós-exercício / Relationship between the peripheral vasodilatory capacity and the hemodynamic mechanisms of post-exercise hypotension
O mecanismo hemodinâmico responsável pela hipotensão pós-exercício aeróbico varia entre os indivíduos, sendo interessante avaliar a possível influência da capacidade de vasodilatação periférica nesses mecanismos. Para tanto, 22 homens normotensos submeteram-se a 2 sessões experimentais: Controle (C - repouso) e Exercício (E- cicloergômetro, 45
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 12/03/2012
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4. Effects of experimental pulmonary hemodynamic overload on cardiovascular system and pulmonary structural and function / Efeitos da sobrecarga hemodinâmica pulmonar experimental no sistema cardiovascular e na estrutura e função pulmonar
INTRODUCTION: The pulmonary hemodynamic overload (PHO) is associates to high mortality and morbidity clinical entities as: postpneumonectomy pulmonary edema, pulmonary thromboembolism, lung transplantation and physiological situations as physical exercise. Some pathophysiological aspects related to PHO are not elucidated. OBJECTIVES: To study pulmonary and c
Publicado em: 2010
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5. Desenvolvimento de metodos opticos para o estudo do acoplamento neuro-vascular-metabolico intrinseco a dinamica cerebral / Development of optical methods to the study of neuro-metabolic-vascular coupling underlying cerebral dynamics
Task-associated neuronal activity leads to a complex chain of biological events within the brain. The increased energetics gives rise to elevated glucose and oxygen consumption in the tissue. Biochemical and neuronal factors induce changes in blood vessels and variations in blood ow, volume and oxygenation. Although it has been extensively investigated, this
Publicado em: 2009
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6. Avaliação dos efeitos nefrotóxicos da associação do tacrolimus (FK 506) e antiinflamatórios não-hormonais em ratos
Introduction: Tacrolimus (FK 506) is a potent immunosuppressive drug that may cause nephrotoxicity decreasing the renal blood flow (RBF) and glomerular filtration rate (GFR). Conventional non-steroidal anti-inflammatory drugs (NSAIDs) may cause nephrotoxicity, interfering with renal hemodynamics and fluid and eletrolyte homeostasis. Recently, new selective C
Publicado em: 2005
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7. Thyroid hormone analog 3,5-diiodothyropropionic acid promotes healthy vasculature in the adult myocardium independent of thyroid effects on cardiac function
Patients with hypothyroidism are at a higher risk for coronary vascular disease. Patients with diabetes and related vascular complications also have an increased incidence of low thyroid function. While thyroid hormones (THs) may be key regulators of a healthy vasculature, potential undesirable side effects hinder their use in the treatment of vascular disor
American Physiological Society.
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8. S-nitrosothiol repletion by an inhaled gas regulates pulmonary function
NO synthases are widely distributed in the lung and are extensively involved in the control of airway and vascular homeostasis. It is recognized, however, that the O2-rich environment of the lung may predispose NO toward toxicity. These Janus faces of NO are manifest in recent clinical trials with inhaled NO gas, which has shown therapeutic benefit in s
The National Academy of Sciences.
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9. Developmental adaptation of the mouse cardiovascular system to elastin haploinsufficiency
Supravalvular aortic stenosis is an autosomal-dominant disease of elastin (Eln) insufficiency caused by loss-of-function mutations or gene deletion. Recently, we have modeled this disease in mice (Eln+/–) and found that Eln haploinsufficiency results in unexpected changes in cardiovascular hemodynamics and arterial wall structure. Eln+/– animals were fou
American Society for Clinical Investigation.
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10. CLINICAL TRIALS OF THE ABDOMINAL LEFT VENTRICULAR ASSIST DEVICE (ALVAD): PROGRESS REPORT
An abdominal left ventricular assist device (ALVAD) is undergoing controlled clinical trials in our institution. The ALVAD is pneumatically-actuated, synchronously or asynchronously with an external console and is interposed between the apex of the left ventricle and the infrarenal abdominal aorta. It is an order of magnitude more effective than conventional
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11. Mobilized bone marrow cells repair the infarcted heart, improving function and survival
Attempts to repair myocardial infarcts by transplanting cardiomyocytes or skeletal myoblasts have failed to reconstitute healthy myocardium and coronary vessels integrated structurally and functionally with the remaining viable portion of the ventricular wall. The recently discovered growth and transdifferentiation potential of primitive bone marrow cells (B
The National Academy of Sciences.
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12. Adaptation to increased dietary salt intake in the rat. Role of endogenous nitric oxide.
Previous studies have suggested that nitric oxide (NO) plays a role in regulation of renal vascular tone and sodium handling. We questioned whether the effects of NO synthase inhibition on renal function are direct or due to increased renal perfusion pressure (RPP) and whether stimulation of endogenous NO activity plays a role in adaptation to increased diet