Full Repair Of The Damage
Mostrando 1-12 de 34 artigos, teses e dissertações.
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1. Função punitiva da indenização por danos extra-patrimoniais e sua compatibilidade no ordenamento jurídico brasileiro
Este trabalho intenta analisar a função punitiva da indenização por danos extra-patrimoniais, aceita pelos tribunais como, ao lado do escopo reparatório, elemento da responsabilidade civil. Para tanto, analisa-se os elementos da responsabilidade civil em sua função reparatória e, posteriormente, se nosso sistema normativo aceita que a responsabilidad
Publicado em: 2010
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2. Responsabilidade civil e a transfusão de sangue / Civil responsibility and blood transfusion
The standardization of homoterapy under the legislation led to a integration of Medicine and Law. With the ease of access to the judicial system, a lot of lawsuits were filed against hospitals and medicine professionals involved in the blood transfusion process seeking reparation of losses due to the activity. On one side are the medicine professionals that
Publicado em: 2010
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3. Condrogenese a partir de celulas Mesenquimais do cordão umbilical humano estimuladas com IGF-1, TGF- beta 3, BMP-6 e BMP-2 em sistema de cultura monolayer e micromass / Chondrogenesis from Human Umbilical Cord Blood Mesenchymal Cells stimulated with TGF-beta3, IGF-1, BMP-2 and BMP-6
O sangue do cordão umbilical contem células tronco mesenquimais (CTM) indiferenciadas que tem potencial condrogênico e podem ser usadas para reparo de lesão articular. Durante o processo de condrogênese, a atuação de fatores de crescimento ainda não está totalmente elucidada. Este estudo teve como objetivo avaliar a formação de condrócitos, matri
Publicado em: 2009
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4. Differential proteomics and physiological characterization of two clones of Coffea canephora under water deficit / Proteômica diferencial e caracterização fisiológica de dois clones de Coffea canephora sob déficit hídrico
Understanding the mechanisms by which plants respond to water stress is crucial to predict the climate change impacts on crop productivity and ecosystems. Plants have long- distance root-to-shoot signaling mechanisms that generate the orchestration of adaptive responses. Root system signaling, as well as shoot, is often considerate important in growth contro
Publicado em: 2008
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5. A Structural Model for the Damage-sensing Complex in Bacterial Nucleotide Excision Repair*
Nucleotide excision repair is distinguished from other DNA repair pathways by its ability to process a wide range of structurally unrelated DNA lesions. In bacteria, damage recognition is achieved by the UvrA·UvrB ensemble. Here, we report the structure of the complex between the interaction domains of UvrA and UvrB. These domains are necessary and suff
American Society for Biochemistry and Molecular Biology.
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6. The Functions of Budding Yeast Sae2 in the DNA Damage Response Require Mec1- and Tel1-Dependent Phosphorylation
DNA damage checkpoint pathways sense DNA lesions and transduce the signals into appropriate biological responses, including cell cycle arrest, induction of transcriptional programs, and modification or activation of repair factors. Here we show that the Saccharomyces cerevisiae Sae2 protein, known to be involved in processing meiotic and mitotic double-stran
American Society for Microbiology.
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7. Complementation of the DNA repair-deficient swi10 mutant of fission yeast by the human ERCC1 gene.
In human cells DNA damage caused by UV light is mainly repaired by the nucleotide excision repair pathway. This mechanism involves dual incisions on both sides of the damage catalyzed by two nucleases. In mammalian cells XPG cleaves 3' of the DNA lesion while the ERCC1-XPF complex makes the 5' incision. The amino acid sequence of the human excision repair pr
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8. A common mutational pattern in Cockayne syndrome patients from xeroderma pigmentosum group G: Implications for a second XPG function
Xeroderma pigmentosum (XP) patients have defects in nucleotide excision repair (NER), the versatile repair pathway that removes UV-induced damage and other bulky DNA adducts. Patients with Cockayne syndrome (CS), another rare sun-sensitive disorder, are specifically defective in the preferential removal of damage from the transcribed strand of active genes,
The National Academy of Sciences of the USA.
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9. Transatrial Closure of Postinfarction Ventricular Septal Defect
While operative approaches to postinfarction ventricular septal defect emphasize repair through the infarcted area, we present a case that illustrates the transatrial approach in an acute setting in which no discrete infarct or other abnormality of the free ventricular wall is apparent. In such a setting, transatrial repair of mid-muscular or posterior defec
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10. Transgenic mice that overexpress the human trefoil peptide pS2 have an increased resistance to intestinal damage.
pS2 is a member of the trefoil peptide family, all of which are overexpressed at sites of gastrointestinal injury. We hypothesized that they are important in stimulating mucosal repair. To test this idea, we have produced a transgenic mice strain that expresses human pS2 (hpS2) specifically within the jejunum and examined the effect of this overexpression on
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11. Requirement of RAD5 and MMS2 for Postreplication Repair of UV-Damaged DNA in Saccharomyces cerevisiae
UV lesions in the template strand block the DNA replication machinery. Genetic studies of the yeast Saccharomyces cerevisiae have indicated the requirement of the Rad6-Rad18 complex, which contains ubiquitin-conjugating and DNA-binding activities, in the error-free and mutagenic modes of damage bypass. Here, we examine the contributions of the REV3, RAD30, R
American Society for Microbiology.
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12. Current state of cartilage tissue engineering
Damage to cartilage is of great clinical consequence given the tissue's limited intrinsic potential for healing. Current treatments for cartilage repair are less than satisfactory, and rarely restore full function or return the tissue to its native normal state. The rapidly emerging field of tissue engineering holds great promise for the generation of functi
BioMed Central.