Adipoq
Mostrando 1-12 de 15 artigos, teses e dissertações.
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1. Pancreatic β-cell function is inhibited by miR-3666 in type 2 diabetes mellitus by targeting adiponectin
Type 2 diabetes mellitus (T2D) is a common endocrine and metabolic disorder, and poses threats to human health worldwide. Recently, microRNAs (miRNAs) have been suggested to play important roles in the pathophysiology of T2D. In this study, we explored the role of miR-3666 in T2D. miR-3666 was significantly down-regulated in the serum of T2D patients when co
Braz J Med Biol Res. Publicado em: 27/05/2019
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2. Adipokine gene variability and plasma levels in patients with chronic periodontitis -a case-control study
Abstract: Specific variants in genes that encode adipokines and their mRNA and protein expression were previously studied in type 2 diabetes mellitus (T2DM) and obesity, and similar studies have been performed for chronic periodontitis (CP). The aim of this case–control study was to investigate the possible impacts of adiponectin (ADIPOQ), leptin (LEP) and
Braz. oral res.. Publicado em: 25/04/2019
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3. Effects of polymorphisms in APOB, APOE, HSD11β1, PLIN4, and ADIPOQ genes on lipid profile and anthropometric variables related to obesity in children and adolescents
Abstract Genes can influence lipid profile and anthropometric variables related to obesity. The present study aimed to verify if variants of the APOE, APOB, ADIPOQ, HSD11β1, and PLIN4 genes are associated with lipid levels or anthropometric variables in a sample comprised of 393 Euro-Brazilian children and adolescents. DNA was genotyped by TaqMan allelic di
Genet. Mol. Biol.. Publicado em: 23/11/2018
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4. Adiponectin promoter polymorphisms are predictors of lipid profile improvement after bariatric surgery
Abstract Our aim was to investigate if single nucleotide polymorphisms (SNPs) located in the 5′ regions of leptin (LEP, -2548 G > A, rs7799039), resistin (RETN, -420 C > G, rs1862513) and adiponectin (ADIPOQ, -11391 G > A, rs17300539 and -11377 C > G, rs266729) genes were related to changes in body mass index (BMI) and metabolic variables after bariatric s
Genet. Mol. Biol.. Publicado em: 23/10/2017
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5. Polymorphisms FTO rs9939609, PPARG rs1801282 and ADIPOQ rs4632532 and rs182052 but not lifestyle are associated with obesity related-traits in Mexican children
Abstract Concerning the genetic factors of obesity, no consistent association between populations has been reported, which may be due to the frequency of polymorphisms, the lifestyle of studied populations and its interaction with other factors. We studied a possible association of polymorphisms FTO rs9939609, PPARG rs1801282, and ADIPOQ rs4632532 and rs1820
Genet. Mol. Biol.. Publicado em: 14/07/2016
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6. Swim training and the genetic expression of adipokines in monosodium glutamate-treated obese rats
Objective The aim of this study was to evaluate the genetic expression of adipokines in the adipocytes of monosodium glutamate (MSG)-treated obese rats submitted to physical activity.Materials and methods Obesity was induced by neonatal MSG administration. Exercised rats (MSG and control) were subjected to swim training for 30 min for 10 weeks, whereas their
Arch. Endocrinol. Metab.. Publicado em: 2015-06
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7. Adiponectina, perfil metabólico e risco cardiovascular em pacientes com síndromes coronarianas agudas / Adiponectin, metabolic profile and cardiovascular risk in patients with acute coronary syndromes
O tecido adiposo é considerado não somente uma fonte de energia estocável, mas principalmente um órgão endócrino que secreta várias citoquinas, as quais podem contribuir para o desenvolvimento de doenças relacionadas à obesidade, incluindo o diabetes mellitus e a doença vascular aterosclerótica. Dentre esse pool de moléculas, a adiponectina (Arcp
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 11/08/2011
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8. Lack of evidence for intermolecular epistatic interactions between adiponectin and resistin gene polymorphisms in Malaysian male subjects
Epistasis (gene-gene interaction) is a ubiquitous component of the genetic architecture of complex traits such as susceptibility to common human diseases. Given the strong negative correlation between circulating adiponectin and resistin levels, the potential intermolecular epistatic interactions between ADIPOQ (SNP+45T > G, SNP+276G > T, SNP+639T > C and SN
Genetics and Molecular Biology. Publicado em: 15/12/2011
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9. ADIPOQ e adiponectina: base comum da hiperglicemia e doença arterial coronariana?
Os níveis plasmáticos de adiponectina e o gene codante desta proteína, ADIPOQ, parecem explicar parte da interação de doenças como obesidade, resistência à insulina, diabetes melito tipo 2 (DM2) e doença arterial coronariana (DAC). Apresentamos as evidências do papel tanto dos níveis de adiponectina quanto das variantes no ADIPOQ na modulação da
Arquivos Brasileiros de Endocrinologia & Metabologia. Publicado em: 2011-10
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10. Adiponectina e baixo risco cardiometabólico em obesas
OBJETIVO: Investigar a associação entre níveis elevados de adiponectina plasmática (AdipoQ) e baixo risco cardiometabólico (RCM) em obesas (MOb). SUJEITOS E MÉTODOS: Estudo caso-controle, pareado pela idade com 306 MOb, IMC > 30 kg/m², sendo 66 (21,6%) casos - todos não hipertensos, normolipídicos e não diabéticos - realizados em ambulatórios ref
Arquivos Brasileiros de Endocrinologia & Metabologia. Publicado em: 2011-03
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11. Fatores associados ao baixo risco cardiometabólico em mulheres obesas
OBJETIVO: Identificar fatores associados ao baixo risco cardiometabólico (RCM) em mulheres obesas (MOb) atendidas em ambulatórios especializados do Sistema Único de Saúde (SUS), Salvador, Bahia, Brasil. SUJEITOS E MÉTODOS: Estudo caso-controle, pareado pela idade com 306 MOb, índice de massa corporal (IMC) > 30 kg/m², sendo 66 (21,6%) casos - todos n�
Arquivos Brasileiros de Endocrinologia & Metabologia. Publicado em: 2010-02
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12. Estudo dos genes TNF alfa, ADIPOQ e STATH entre portadores de fibrose cistica / Modifiers genes : TNF alfa, ADIPOQ and STATH in cystic fibrosis patients from Campinas
A Fibrose Cística (FC) possui uma grande variabilidade de expressão fenotípica, o que significa que crianças com o mesmo genótipo podem diferir quanto à sua apresentação. A proteína defeituosa formada é chamada CFTR (proteína reguladora da conductância iônica), causa transporte anormal de sódio e cloro através da membrana apical das células e
Publicado em: 2009