Sulfonamides
Mostrando 25-36 de 203 artigos, teses e dissertações.
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25. Estudio termodinámico de la transferencia de acetaminofén desde el agua hasta el octanol
The partition coefficients and solubilities in octanol, water and mutually saturated octanol-water phases were determined for acetaminophen at 25.0, 30.0, 35.0, and 40.0 °C. By means of Gibbs and van't Hoff thermodynamic analyses it may observe that the transfer of this drug from water to octanol is spontaneous and mainly driven enthalpically. As in other s
Revista Brasileira de Ciências Farmacêuticas. Publicado em: 2004-09
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26. Thermodynamic study of the solubility of some sulfonamides in cyclohexane
As variações nas funções termodinâmicas energia de Gibbs, entalpia e entropia para os processos de solução de uma série de sulfonamidas em cicloexano foram determinadas nas temperaturas de 20,0 a 40,0 °C. A energia de Gibbs de excesso e os coeficientes de atividade destes solutos também foram calculados. Estes resultados são discutidos em termos d
Journal of the Brazilian Chemical Society. Publicado em: 2003-10
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27. A randomized, pilot trial comparing full versus escalating dose regimens for the desensitization of AIDS patients allergic to sulfonamides
Sulfonamides are drugs extensively used in the management of AIDS patients. However, the use of sulfonamides is often associated with the development of allergic reactions, provoking the substitution of the drug (by another that may be less effective); alternatively attempts are made to desensitize the patient. OBJECTIVE: Compare two drug regimens (full vs.
Brazilian Journal of Infectious Diseases. Publicado em: 2002-12
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28. Studies on the Action of Sulfonamides on the Respiration and Growth of Bacteria 1: A. Factors Controlling the Inhibition by Sulfonamides of Carboxylases. III. Antagonism Between Neopeptone and Serum Proteins, and Sulfonamides
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29. Synergism of Polymyxin and Sulfonamides in L-forms of Staphylococcus aureus and Proteus mirabilis
Earlier workers suggested that synergism of polymyxin and sulfonamides occurred because sulfonamides produced changes in the cell wall which permitted the more ready access of polymyxin. We tested this hypothesis by using L-forms, which lack cell wall. The demonstration that synergism of polymyxin and sulfonamides occurs in L-forms of Proteus mirabilis and S
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30. Inhibition by sulfonamides of the candidacidal activity of human neutrophils
Sulfonamides reduced substantially the ability of normal human neutrophils to kill strains of Candida albicans and Candida tropicalis, and impaired to a lesser extent their activity against Staphylococcus aureus 502A and Serratia marcescens. Sulfonamides also inhibited (a) iodination of Candida cells by normal neutrophils; (b) candidacidal activity in cell-f
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31. Activity of Brodimoprim and Metioprim Alone and in Combination with Sulfonamides Against Anaerobic Bacteria
The activities of the 2,4-diamino-5-benzylpyrimidines brodimoprim and metioprim against anaerobic bacteria were tested alone and in combination with sulfonamides. Alone they were two to four times more active than trimethoprim, but in combination with sulfonamides, their activity was slightly lower than that of trimethoprim plus sulfamethoxazole.
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32. Studies on the Action of Sulfonamides on the Respiration and Growth of Bacteria 1: A. Factors Controlling the Inhibition by Sulfonamides of Carboxylases. I. Antagonism Between Cocarboxylase and Sulfathiazole
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33. Studies on the Action of Sulfonamides on the Respiration and Growth of Bacteria 1: A. Factors Controlling the Inhibition by Sulfonamides of Carboxylases. II. Antagonism Between p-Aminobenzoic Acid and Sulfathiazole
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34. Characterization of mutationally altered dihydropteroate synthase and its ability to form a sulfonamide-containing dihydrofolate analog.
Among spontaneous mutants of Escherichia coli selected for resistance against sulfonamides, thermosensitive strains were found. These were shown to possess a changed dihydropteroate synthase (EC 2.5.1.15), which had a substantially higher Km value for its normal substrate, p-aminobenzoic acid, and an about 150-fold higher Km for sulfonamides. The mutationall
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35. The Mechanism of Resistance to Sulfonamides: II. Absence of Correlation between Resistance and the Formation of Arylamine by Staphylococcus aureus. Noninterference with the Utilization of Glucose as a Critical Factor in the Development of Resistance to Sulfonamides1
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36. The Mechanism of Resistance to Sulfonamides: III. Pantothenic Acid and Tryptophane Metabolism: The Role of Pantothenic Acid in the Synthesis of Tryptophane by Staphylococcus aureus and the Effect of Vitamins on Tryptophane in Exercising Antagonism to Sulfonamides1