Sodium Appetite
Mostrando 1-12 de 24 artigos, teses e dissertações.
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1. Changes in sodium appetite evoked by lesions of the commissural nucleus of the tractus solitarius
Ablation of the area postrema/caudal nucleus of the tractus solitarius (NTS) complex increases sodium intake, but the effect of selective lesions of the caudal NTS is not known. We measured depletion-induced sodium intake in rats with electrolytic lesions of the commissural NTS that spared the area postrema. One day after the lesion, rats were depleted of so
Brazilian Journal of Medical and Biological Research. Publicado em: 2009-06
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2. Núcleo central da amígdala e Núcleo parabraquial lateral no controle da ingestão de sódio
Previous studies have shown the importance of serotonergic GABAergic and aadrenergic mechanisms of the lateral parabraquial nucleus (LBPN) in the control of sodium intake. The importance of the central nucleus of the amygdala (CeA) for sodium intake induced by different protocols was also demonstrated. Considering the studies showing reciprocal connections b
Publicado em: 2009
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3. Aldosterona e apetite ao sódio: uma nova evidência para um antigo efeito / Aldosterone and sodium appetite: a new evidence for an old effect
A aldosterona é um hormônio mineralocorticóide que atua no sistema nervoso central (SNC) para estimular o apetite ao sódio, no entanto, pouco se sabe sobre o mecanismo central envolvido nesta resposta. Recentemente, estudos neuroanatômicos identificaram um grupo de neurônios sensíveis a aldosterona, localizados exclusivamente nos núcleos do trato sol
Publicado em: 2009
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4. Role of the serotoninergic system in the sodium appetite control
Este artigo revisa o papel do sistema serotoninérgico no controle do apetite ao sódio. Dados derivados da administração periférica e icv de agentes serotoninérgicos demonstraram a participação de receptores 5-HT2/3 na modulação do apetite ao sódio. Estas observações foram estendidas com os estudos realizados após a depleção cerebral de seroto
Anais da Academia Brasileira de Ciências. Publicado em: 2007-06
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5. Water deprivation and the double- depletion hypothesis: common neural mechanisms underlie thirst and salt appetite
Water deprivation-induced thirst is explained by the double-depletion hypothesis, which predicts that dehydration of the two major body fluid compartments, the extracellular and intracellular compartments, activates signals that combine centrally to induce water intake. However, sodium appetite is also elicited by water deprivation. In this brief review, we
Brazilian Journal of Medical and Biological Research. Publicado em: 05/04/2007
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6. Mineralocorticoid modulation of central angiotensin-induced neuronal activity, water intake and sodium appetite
Central angiotensin II (AngII) stimulates water and salt solution intake. Pretreatment with low-dose mineralocorticoid (DOCA) enhances this AngII-induced intake of salt solutions (the synergy theory) in Wistar and Sprague Dawley rats but not in Fischer rats. This response is mediated via the AT-1 receptor. Electrophysiological experiments using iontophoretic
Brazilian Journal of Medical and Biological Research. Publicado em: 26/03/2007
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7. Matching salt intake to physiological need in rats using foraging protocols
Several studies of the quantitative relationship between sodium need and sodium intake in rats are reviewed. Using acute diuretic treatment 24 h beforehand, intake matches need fairly accurately when intake is spread out in time by using a hypotonic solution of NaCl. In contrast, using a hypertonic solution, intake is typically double the need. Using the sam
Brazilian Journal of Medical and Biological Research. Publicado em: 14/03/2007
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8. Sodium appetite specificity: a possible hormonal contribution. / Especificidade do apetite ao sódio: uma possível contribuição hormonal.
A hipótese do sinergismo entre dois hormônios responsáveis pela conservação de sódio, a aldosterona e a ANG II, explica uma característica do apetite ao sódio, a ingestão de NaCl hipertônico em um animal hipovolêmico. Ingestão de NaCl hipertônico pode ser induzida em ratos normovolêmicos que receberam um tratamento combinado de mineralocorticó
Publicado em: 2006
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9. Chronic excitotoxic lesion of the dorsal raphe nucleus induces sodium appetite
We determined if the dorsal raphe nucleus (DRN) exerts tonic control of basal and stimulated sodium and water intake. Male Wistar rats weighing 300-350 g were microinjected with phosphate buffer (PB-DRN, N = 11) or 1 µg/0.2 µl, in a single dose, ibotenic acid (IBO-DRN, N = 9 to 10) through a guide cannula into the DRN and were observed for 21 days in order
Brazilian Journal of Medical and Biological Research. Publicado em: 2005-11
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10. Brain serotonin depletion enhances the sodium appetite induced by sodium depletion or beta-adrenergic stimulation
Objetivamos avaliar a influência da depleção cerebral de serotonina no apetite por sódio. Ratos depletados de serotonina através da administração sistêmica de paraclorofenilalanina (300 mg/kg, ip, por 2 dias) exibiram intensa resposta natriorexigênica induzida pela depleção de sódio (furosemida, 20 mg/kg, sc, 24 h antes da oferta de água e NaCl
Anais da Academia Brasileira de Ciências. Publicado em: 2004-03
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11. Effect of electrolytic lesion of the dorsal raphe nucleus on water intake and sodium appetite
The present study determined the effect of an electrolytic lesion of the dorsal raphe nucleus (DRN) on water intake and sodium appetite. Male Wistar rats weighing 290-320 g with a lesion of the DRN (L-DRN), performed two days before experiments and confirmed by histology at the end of the experiments, presented increased sensitivity to the dehydration induce
Brazilian Journal of Medical and Biological Research. Publicado em: 2003-12
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12. Interaction between paraventricular nucleus and septal area in the control of physiological responses induced by angiotensin II
We determined the effects of losartan (40 nmol) and PD 123319 (40 nmol) (both non-peptides and selective antagonists of the AT1 and AT2 angiotensin receptors, respectively), and [Sar¹, Ala8] angiotensin II (ANG II) (40 nmol) (a non-selective peptide antagonist of angiotensin receptors) injected into the paraventricular nucleus (PVN) on the water and salt ap
Brazilian Journal of Medical and Biological Research. Publicado em: 2002-09