Renovascular Hypertension
Mostrando 25-36 de 61 artigos, teses e dissertações.
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25. Oral administration of L-arginine decreases blood pressure and increases renal excretion of sodium and water in renovascular hypertensive rats
The two-kidney, one-clip renovascular (2K1C) hypertension model is characterized by a reduction in renal flow on the clipped artery that activates the renin-angiotensin system. Endothelium dysfunction, including diminished nitric oxide production, is also believed to play a role in the pathophysiology of this model. Some studies have shown an effect of L-arg
Brazilian Journal of Medical and Biological Research. Publicado em: 2003-07
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26. Abnormal proliferative response of the carotid artery of spontaneously hypertensive rats after angioplasty may be related to the depolarized state of its smooth muscle cells
Hypertension is one of the major precursors of atherosclerotic vascular disease, and vascular smooth muscle abnormal cell replication is a key feature of plaque formation. The present study was conducted to examine the relationship between hypertension and smooth muscle cell proliferation after balloon injury and to correlate neointima formation with resting
Brazilian Journal of Medical and Biological Research. Publicado em: 2000-08
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27. Renovascular hypertension commencing during fetal life
We report three infants with severe, early hypertension due to unilateral renovascular disease, whose cardiovascular changes, or polycythaemia, or both, indicated they had been affected as fetuses. All underwent unilateral nephrectomy, and had a similar histology, with patchy areas having relatively normal glomeruli but immature proximal tubules. This patter
BMJ Group.
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28. Decreased susceptibility to renovascular hypertension in mice lacking the prostaglandin I2 receptor IP
Persistent reduction of renal perfusion pressure induces renovascular hypertension by activating the renin-angiotensin-aldosterone system; however, the sensing mechanism remains elusive. Here we investigated the role of PGI2 in renovascular hypertension in vivo, employing mice lacking the PGI2 receptor (IP–/– mice). In WT mice with a two-kidney, one-clip
American Society for Clinical Investigation.
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29. Angiotensin II-noradrenergic interactions in renovascular hypertensive rats.
This study tested the hypothesis that interactions of endogenous angiotensin II (AII) with the noradrenergic neuroeffector junction are important in renin-dependent hypertension. In the in situ blood-perfused rat mesentery, in normal rats exogenous AII potentiated mesenteric vascular responses to periarterial (sympathetic) nerve stimulation (PNS) more than v
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30. Chronic caffeine administration exacerbates renovascular, but not genetic, hypertension in rats.
The purpose of this study was to determine whether or not caffeine would exacerbate renovascular hypertension. Therefore, we examined the effects of chronic caffeine administration on arterial blood pressure in rats subjected to either unilateral renal artery clipping (2K-1C rats) or sham-operation. Animals in each group were randomly assigned to receive eit
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31. Renovascular Hypertension
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32. Anticardiolipin antibodies and renovascular hypertension.
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33. Late results after surgical treatment of renovascular hypertension. A follow-up study of 122 patients 2-18 years after surgery.
During the period 1963-1980, 122 patients were operated on for renovascular hypertension at surgical department D, vascular section, Rigshospitalet, Copenhagen. Seventeen patients, with a median age of 24 years, had fibromuscular hyperplasia and 95 patients, with a median age of 48 years, had atherosclerosis. Twenty-four of the latter had bilateral renal art
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34. Plasma atrial natriuretic factor concentrations in essential and renovascular hypertension.
Plasma atrial natriuretic factor concentrations were measured in 44 patients with mild untreated essential hypertension and 48 normotensive controls. Mean venous plasma atrial natriuretic factor concentrations were 13.2 (SEM 1.5) and 13.0 (1.3) ng/l in the hypertensive patients and controls, respectively. Plasma atrial natriuretic factor concentrations were
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35. Captopril Renograms for Detecting Renovascular Hypertension
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36. RENAL AUTOTRANSPLANTATION FOR RENOVASCULAR HYPERTENSION: REPORT OF FOUR CASES*