Protein Losing Enteropathy
Mostrando 1-12 de 22 artigos, teses e dissertações.
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1. Outcomes of the Conversion of the Fontan-Kreutzer Operation to a Total Cavopulmonary Connection for the Failing Univentricular Circulation
Resumo Fundamento: O procedimento de Fontan-Kreutzer (FK) foi amplamente realizado no passado, mas a longo prazo gerou muitas complicações, resultando em falha na circulação univentricular. A conversão para conexão cavopulmonar total (CCPT) é uma das opções de tratamento. Objetivo: Avaliar os resultados da conversão de FK para CCPT. Métodos: Re
Arq. Bras. Cardiol.. Publicado em: 2019-02
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2. Protein-losing enteropathy in paracoccidioidomycosis identified by scintigraphy: report of three cases
Paracoccidioidomycosis (PCM) or South American Blastomycosis is a systemic fungal infection caused by a dimorphic fungus, Paracoccidioides brasiliensis. It represents the most important systemic mycosis in South America and with higher prevalence among male inhabitants of the rural area. PCM usually affects the lungs, and rarely the intestines. The authors r
Brazilian Journal of Infectious Diseases. Publicado em: 2010-10
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3. Linfangiectasia intestinal e linfagite lipogranulomatosa em dois caninos / Intestinal lymphangiectasia with lipogranulomatous lymphangitis in two dogs
Relatam-se dois casos adicionais de linfangiectasia intestinal e linfangite lipogranulomatosa (LI/LL) em caninos. Sinais clínicos manifestados pelos animais afetados incluíam diarréia crônica, perda de peso, dificuldade respiratória e anorexia. À necropsia, a mucosa do intestino delgado apresentava-se pálida, macia e com aspecto aveludado mais evident
Publicado em: 2010
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4. Association of protein-losing enteropathy caused by eosinophilic gastroenteritis with essential thrombocytosis: case report
Clinics. Publicado em: 2006-06
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5. Late Clinical Outcomes of the Fontan Operation in Patients with Tricuspid Atresia
OBJECTIVE: Evaluation of the long-term clinical results of the Fontan operation in patients with tricuspid atresia. METHODS: A retrospective analysis was made at the Instituto de Cardiologia do Rio Grande do Sul (Institute of Cardiology of Rio Grande do Sul), from August 1980 through January 2000, of 25 patients with a long-term follow-up, out of a series of
Arquivos Brasileiros de Cardiologia. Publicado em: 2002-07
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6. Protein losing enteropathy associated with collagen diseases.
Four patients with collagen diseases are described, who developed protein losing enteropathy in the course of their disease. Their protein losing enteropathies subsided after treatment with 60 mg/day oral prednisolone. Immunohistological studies of the small intestine showed deposits of C3 in the capillary walls of villi in two patients. Increased capillary
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7. Regression of post-Fontan protein-losing enteropathy. After surgical correction of hemodynamic faults other than high right atrial pressure.
We describe a case of protein-losing enteropathy after a Fontan operation, in the absence of high right atrial pressure. Although partial regression of protein-losing enteropathy was obtained with high doses of cortisone, complete resolution of the phenomenon was accomplished only after surgical closure of a left-to-right shunt through the pulmonary valve.
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8. Correction: Protein-losing enteropathy in patients with Fontan circulation: is it triggered by infection?
A correction to Protein-losing enteropathy in patients with Fontan circulation: is it triggered by infection? by Dominik Lenz, Jorg Hambsch, Peter Schneider, Hans-Jungen Hausler, Ursula Sauer, John Hess and Attila Tarnok.
BioMed Central.
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9. Protein-losing enteropathy caused by Lawsonia intracellularis in a weanling foal
A 5-month-old Morgan filly was presented to the Atlantic Veterinary College with a history of lethargy, fever, depression, anorexia, and dependent ventral edema. Diagnostic tests revealed severe inflammation, hypoproteinemia, and thickened small intestinal loops. Protein-losing enteropathy caused by Lawsonia intracellularis was diagnosed and treated successf
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10. Protein-losing enteropathy
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11. Recurrent pleural effusion, protein-losing enteropathy, malabsorption, and mosaic warts associated with generalized lymphatic hypoplasia
A patient is described who presented with pleural effusions, asymmetrical oedema of the face and limbs, and numerous warts of the hands and feet. He was shown to have vitamin B12 malabsorption, steatorrhoea, and a protein-losing enteropathy. Lymphography showed asymmetrical lymphatic hypoplasia. Jejunal biopsy showed intestinal lymphangiectasia. The hypoprot
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12. Giant inflammatory polyposis in ulcerative colitis presenting with protein losing enteropathy.