Pole Placement
Mostrando 13-24 de 26 artigos, teses e dissertações.
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13. Controle H2/H Infinito de estruturas flexiveis atraves de desigualdades matriciais lineares com alocação de polos / H2/H "Infinity control of flexible strructures through linear matrix inequalities with pole placement
o objetivo deste trabalho é aplicar o controle H2/H Infinito usando desigualdades matriciais lineares com restrições de alocação de pólos em estruturas flexíveis. O problema de controle H2/H Infinito é uma técnica usada para a obtenção de controladores com as propriedades do controle norma H2, que trás desempenho ótimo, e do controle norma H Inf
Publicado em: 2005
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14. Uma proposta de um controlador adaptativo por posicionamento de pólos e estrutura variável
Existem dois métodos principais para a construção de controladores adaptativos. Um deles é o controle adaptativo por modelo de referência (MRAC) e o outro é o controle adaptativo por posicionamento de pólos (APPC). No MRAC, um modelo de referência é escolhido para gerar a trajetória desejada que o sinal de saída da planta deve seguir, e ele pode r
Publicado em: 2005
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15. O algoritmo auto-ajustavel no controle de processos com atraso de transporte variante no tempo
For many years there has been an Interest on controlJers that automatically update their own parameters when controlling compIex processes or when the operation conditions vary. This interest has increased the number of works in adaptive controI area. The generalized minimum variance controller (GMV) is included in these works and it uses the seIf-tuning app
Publicado em: 1992
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16. Division site selection in Escherichia coli involves dynamic redistribution of Min proteins within coiled structures that extend between the two cell poles
The MinCDE proteins of Escherichia coli are required for proper placement of the division septum at midcell. The site selection process requires the rapid oscillatory redistribution of the proteins from pole to pole. We report that the three Min proteins are organized into extended membrane-associated coiled structures that wind around the cell between t
National Academy of Sciences.
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17. The MinE ring required for proper placement of the division site is a mobile structure that changes its cellular location during the Escherichia coli division cycle
Placement of the division site at midcell in Escherichia coli requires the MinE protein. MinE acts by imparting topological specificity to the MinCD division inhibitor, preventing the inhibitor from acting at the midcell site while permitting it to block division at other unwanted sites along the length of the cell. It was previously shown that MinE ass
The National Academy of Sciences.
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18. Division site placement in E.coli: mutations that prevent formation of the MinE ring lead to loss of the normal midcell arrest of growth of polar MinD membrane domains
The MinE protein functions as a topological specificity factor in determining the site of septal placement in Escherichia coli. MinE assembles into a membrane-associated ring structure near midcell and directs the localization of MinD and MinC into a membrane- associated polar zone that undergoes a characteristic pole-to-pole oscillation cycle. Single (green
Oxford University Press.
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19. The N Terminus of MinD Contains Determinants Which Affect Its Dynamic Localization and Enzymatic Activity
MinD is involved in regulating the proper placement of the cytokinetic machinery in some bacteria, including Neisseria gonorrhoeae and Escherichia coli. Stimulation of the ATPase activity of MinD by MinE has been proposed to induce dynamic, pole-to-pole oscillations of MinD in E. coli. Here, we investigated the effects of deleting or mutating conserved resid
American Society for Microbiology.
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20. Rapid pole-to-pole oscillation of a protein required for directing division to the middle of Escherichia coli
Accurate placement of the division septum at the midpoint of Escherichia coli cells requires the combined action of a general division inhibitor (MinC), a site-specific suppressor of division inhibition (MinE), and an ATPase (MinD) that is required for proper functioning of both MinC and MinE. We previously showed that a functional MinE-Gfp fusion accumulate
The National Academy of Sciences.
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21. Coupling of Asymmetric Division to Polar Placement of Replication Origin Regions in Bacillus subtilis
Entry into sporulation in Bacillus subtilis is characterized by the formation of a polar septum, which asymmetrically divides the developing cell into forespore (the smaller cell) and mother cell compartments, and by migration of replication origin regions to extreme opposite poles of the cell. Here we show that polar septation is closely correlated with mov
American Society for Microbiology.
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22. ATP-Dependent Interactions between Escherichia coli Min Proteins and the Phospholipid Membrane In Vitro
Proper placement of the division apparatus in Escherichia coli requires pole-to-pole oscillation of the MinC division inhibitor. MinC dynamics involves a membrane association-dissociation cycle that is driven by the activities of the MinD ATPase and the MinE topological specificity factor, which themselves undergo coupled oscillatory localization cycles. To
American Society for Microbiology.
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23. A gain-of-function mutation in IAA18 alters Arabidopsis embryonic apical patterning
Lateral organ emergence in plant embryos and meristems depends on spatially coordinated auxin transport and auxin response. Here, we report the gain-of-function iaa18-1 mutation in Arabidopsis, which stabilizes the Aux/IAA protein IAA18 and causes aberrant cotyledon placement in embryos. IAA18 was expressed in the apical domain of globular stage embryos
Company of Biologists.
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24. Mechanism controlling perpendicular alignment of the spindle to the axis of cell division in fission yeast
In animal cells, the mitotic spindle is aligned perpendicular to the axis of cell division. This ensures that sister chromatids are separated to opposite sides of the cytokinetic actomyosin ring (CAR). We show that, in fission yeast, spindle rotation is dependent on the interaction of astral microtubules with the cortical actin cytoskeleton. Interaction init