Oncogenic Viruses
Mostrando 13-24 de 213 artigos, teses e dissertações.
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13. Counter-Immunoelectrophoresis: Rapid Method for Detecting Group-Specific Antigen and Antibodies Associated with Oncogenic Ribonucleic Acid Viruses
Counter-immunoelectrophoresis was used for the quantitation of group-specific antigen and antibodies of C-type ribonucleic acid leukemia and sarcoma viruses.
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14. Enhancement of Friend Leukemia Virus Infection in Mice by Guaroa Virus: Quantitation and Action of Various Oncogenic and Nononcogenic Viruses
Previous studies have indicated that Guaroa virus (GV), an arbovirus, enhanced the replication of Friend leukemia virus (FLV) in mice. To study further the interaction of GV and FLV, different levels of FLV activity were inoculated into mice. At a level of activity capable of producing limited splenic response, coinfection with GV resulted in a marked increa
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15. PRODUCTION OF “TUMOR-SPECIFIC” ANTIGENS BY ONCOGENIC VIRUSES DURING ACUTE CYTOLYTIC INFECTIONS
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16. Enhancer activity correlates with the oncogenic potential of avian retroviruses.
Avian retroviruses lacking an oncogene, such as Rous-associated virus 1 (RAV-1), RAV-2, and td mutants of Rous sarcoma virus (RSV), can nevertheless cause leukemias and other neoplastic diseases. During this process, viral DNA integrates near a cellular proto-oncogene, such as c-myc, and thus de-regulates its expression. The virus RAV-0, on the other hand, i
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17. Regions containing repeating base-pairs in DNA from some oncogenic and nononcogenic animal viruses.
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18. Infection of muscle cultures from various species with oncogenic DNA viruses (SV40 and polyoma).
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19. Transformation of Murine Cells by Two “Slow Viruses,” Visna Virus and Progressive Pneumonia Virus
Visna and progressive pneumonia virus (PPV), two antigenically related, non-oncogenic “slow viruses” which have ribonucleic acid (RNA)-dependent deoxyribonucleic acid (DNA) polymerase activity, were examined for their ability to transform cells. Murine cells which had been exposed to either visna or PPV developed foci of altered, spindle-shaped cells 3 t
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20. MECHANISM OF VIRAL CARCINOGENESIS BY DNA MAMMALIAN VIRUSES, II. VIRAL-SPECIFIC RNA IN TUMOR CELLS INDUCED BY “WEAKLY” ONCOGENIC HUMAN ADENOVIRUSES*
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21. Avian reticuloendotheliosis viruses: evolutionary linkage with mammalian type C retroviruses.
Reticuloendotheliosis viruses have been shown to be causative of tumors in a variety of avian species. The major structural protein of these non-genetically transmitted viruses is demonstrated to possess antigenic determinants common to those of all known mammalian type C viruses. These findings establish a mammalian origin for this oncogenic avian retroviru
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22. Influence of enhancer sequences on thymotropism and leukemogenicity of mink cell focus-forming viruses.
Oncogenic mink cell focus-forming (MCF) viruses, such as MCF 247, show a positive correlation between the ability to replicate efficiently in the thymus and a leukemogenic phenotype. Other MCF viruses, such as MCF 30-2, replicate to high titers in thymocytes and do not accelerate the onset of leukemia. We used these two MCF viruses with different biological
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23. Molecular Basis for the Attack on Cancer
Studies of cell transformation by RNA and DNA tumor viruses by DNA-RNA hybridization and extension of these techniques to analysis of human cancer tissue are reviewed. The molecular hybridization technique, it is suggested, will be useful in further studies of the role of oncogenic RNA and/or DNA viruses in causation of cancer.
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24. Comparative studies of interferon and three antiviral agents on neurotropic and oncogenic herpesviruses.
The antiviral effects of four compounds, phosphonoacetate, phosphonoformate, acycloguanosine, and purified human lymphoblastic interferon, were tested against two neurotropic herpesviruses, herpesvirus platyrrhine and herpes simplex virus, and two oncogenic herpesviruses, herpesvirus saimiri and herpesvirus ateles. All four compounds induced different degree