Neurohumoral Activation
Mostrando 1-12 de 12 artigos, teses e dissertações.
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1. Efeitos do treinamento físico aeróbico sobre o remodelamento do ventrículo esquerdo e sua correlação com a ativação neuro-humoral em pacientes com infarto agudo do miocárdio / Effects of aerobic exercise training on left ventricular remodeling and its correlation with neurohumoral activation in patients with acute myocardial infarction.
A literatura mostra um número substancial de trabalhos que descrevem a influência do treinamento físico sobre o remodelamento ventricular em pacientes no contexto do pós-infarto agudo do miocárdio (IAM). Entretanto, essas publicações têm apresentado resultados conflitantes. O presente estudo teve como objetivo avaliar os efeitos do treinamento físic
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 13/04/2012
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2. Remodeling in the ischemic heart: the stepwise progression for heart
Abstract Coronary artery disease is the leading cause of death in the developed world and in developing countries. Acute mortality from acute myocardial infarction (MI) has decreased in the last decades. However, the incidence of heart failure (HF) in patients with healed infarcted areas is increasing. Therefore, HF prevention is a major challenge to the hea
Brazilian Journal of Medical and Biological Research. Publicado em: 2011-09
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3. Neurohumoral activation in heart failure: the role of adrenergic receptors
A insuficiência cardíaca (IC) é a via final comum da maioria das doenças cardiovasculares e uma das maiores causas de morbi-mortalidade. O desenvolvimento do estágio final da IC freqüentemente envolve um insulto inicial do miocárdio, reduzindo o débito cardíaco e levando ao aumento compensatório da atividade do sistema nervoso simpático (SNS). Exi
Anais da Academia Brasileira de Ciências. Publicado em: 2006-09
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4. Role of hepatic autoregulation in defense against hypoglycemia in humans.
To assess the role of hepatic autoregulation in defense against hypoglycemia, we compared the effects of complete blockade of glucose counterregulation with those of blockade of only neurohumoral counterregulation during moderate (approximately 50 mg/dl) and severe (approximately 30 mg/dl) hypoglycemia induced by physiologic hyperinsulinemia during subcutane
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5. Heart failure and atrial fibrillation: current concepts and controversies.
Heart failure and atrial fibrillation are very common, particularly in the elderly. Owing to common risk factors both disorders are often present in the same patient. In addition, there is increasing evidence of a complex, reciprocal relation between heart failure and atrial fibrillation. Thus heart failure may cause atrial fibrillation, with electromechanic
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6. Neurohumoral stimulation in type-2-diabetes as an emerging disease concept
Neurohumoral stimulation comprising both autonomic-nervous-system dysfunction and activation of hormonal systems including the renin-angiotensin-aldosterone system (RAAS) was found to be associated with Type-2-diabetes (T2D). Therapeutic strategies such as RAAS interference proved to be beneficial in both T2D treatment and prevention. In addition to an activ
BioMed Central.
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7. Brain nuclear factor-kappa B activation contributes to neurohumoral excitation in angiotensin II-induced hypertension
Oxford University Press.
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8. Apparent paradox of neurohumoral axis inhibition after body fluid volume depletion in patients with chronic congestive heart failure and water retention.
BACKGROUND--Hypovolaemia stimulates the sympathoadrenal and renin systems and water retention. It has been proposed that in congestive heart failure reduction of cardiac output and any associated decrease in blood pressure cause underfilling of the arterial compartment, which promotes and perpetuates neurohumoral activation and the retention of fluid. This s
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9. Heart rate variability in left ventricular dysfunction and heart failure: effects and implications of drug treatment.
OBJECTIVE--To review the importance of heart rate variability analysis in left ventricular dysfunction and heart failure and to assess the effects of drug treatment. In patients with left ventricular dysfunction or heart failure, a low heart rate variability is a strong predictor of a low probability of survival. Because drug treatment in these patients has
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10. A functional role for endogenous atrial natriuretic peptide in a canine model of early left ventricular dysfunction.
Asymptomatic or early left ventricular dysfunction in humans is characterized by increases in circulating atrial natriuretic peptide (ANP) without activation of the renin-angiotensin-aldosterone system (RAAS). We previously reported a canine model of early left ventricular dysfunction (ELVD) produced by rapid ventricular pacing and characterized by an identi
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11. Angiotensin converting enzyme inhibition modulates endogenous endothelin in chronic canine thoracic inferior vena caval constriction.
Endothelin (ET) is a potent vasoconstrictor peptide which is elevated in plasma in congestive heart failure. Recent studies suggest an important role for angiotensin II (AII) in the activation of ET in cultured cardiomyocytes. Chronic thoracic inferior vena caval constriction (TIVCC) is a model of reduced cardiac output that mimics the neurohumoral activatio
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12. Augmented agonist-induced Ca(2+)-sensitization of coronary artery contraction in genetically hypertensive rats. Evidence for altered signal transduction in the coronary smooth muscle cells.
The Ca2+ responsiveness of vascular smooth muscle myofilaments is not unique: it is increased during neuro-humoral activation and decreased during beta-adrenergic stimulation. In this study we tested whether an augmented Ca2+ responsiveness of smooth muscle myofilaments may contribute to the increased coronary tone observed in hypertension using beta-escin-p