Nerve Conduction Velocity
Mostrando 13-24 de 234 artigos, teses e dissertações.
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13. Influence of dietary myoinositol on nerve conduction and inositol phospholipids in normal and diabetic rats.
Observations have been made on motor conduction velocity in the tibial nerve of rats given 35% myoinositol in the diet. Comparison between the values before and with up to nine weeks of dosing revealed no alteration in conduction velocity. In such animals, the free myoinositol content in the sciatic nerve was increased; there was no detectable alteration in
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14. Neurophysiological studies of autogenous sural nerve grafts.
Sixteen autogenous sural nerve grafts used for ulnar and median injuries in the forearm have been studied neurophysiologically up to two and a half years after operation. Motor and sensory nerve conduction studies revealed a slow but sustained improvement during the follow-up period. By two years, motor conduction velocity across the graft itself reached in
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15. Improved estimates of conduction velocity distributions using single unit action potentials.
Single unit potentials were recorded from sural and medial gastrocnemius nerves. Action potential amplitude, integrated area and half-width (duration) were approximately proportional to conduction velocity, raised to the powers 1.5, 1 and -0.5 respectively with the sural nerve, and 2, 1.5 and -0.5 for the medial gastrocnemius nerve. These empirical relations
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16. Nerve conduction velocity in experimental diabetes in the rat and rabbit.
Tibial motor nerve conduction velocity was measured in rats, before and two months after the induction of diabetes with streptozotocin. A second group of diabetic animals was also administered 1% dietary myoinositol supplements. An analysis of variance was performed on these data. Myoinositol supplements had no effect whatsoever. The period of diabetes had a
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17. Changes in nerve function and nerve fibre structure induced by acute, graded compression.
Rabbit tibial nerves were subjected to direct, acute graded compression by means of an inflatable compression chamber. The acute and long term effects of 50, 200 and 400 mmHg applied for two hours on nerve function and nerve fibre structure were investigated. A pressure of 50 mmHg applied for two hours induced only minimal or no acute deterioration of maxima
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18. Conduction velocity in the proximal segments of a motor nerve in the Guillain-Barré syndrome.
Conduction velocity from spinal cord to axilla (estimated using the F wave) has been compared with conduction velocity from axilla to wrist (measured in the conventional manner) in the motor fibres of the ulnar nerve in 17 controls subjects and in 11 patients with the Guillain-Barré syndrome (GBS). In the patients with GBS the conduction velocity was, in ge
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19. Measurement of motor conduction velocity with Hopf's technique in the diagnosis of mild peripheral neuropathies
Hopf's technique was used to measure motor nerve conduction velocity of fibres in the deep peroneal nerve in clinical conditions where damage to the peripheral nervous system was probable. A particular distribution of motor nerve fibre conduction velocity was shown by the analysis of the findings in some of the groups investigated, illustrating the advantage
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20. The conduction velocity of slower and the fastest fibres in infancy and childhood.
Motor nerve conduction velocity of the ulnar nerve was measured in 54 infants of various ages. Conduction velocity of slower fibres was measured using Hopf's technique, and maximal conduction velocity was measured with the usual method. Both maximal conduction velocity and conduction velocity of slower fibres increased with maturation. The former was about 6
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21. Conduction velocity along human nociceptive reflex afferent nerve fibres.
The conduction velocity along the nociceptive flexor reflex afferent nerve fibres was investigated in human subjects. The posterior tibal nerve was stimulated at two sites by single painful electrical shocks of 1.0 ms duration and with adequate intensity and the reflex EMG discharges were recorded from the short head of the biceps femoris muscle. The fastest
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22. Abnormalities in the vagus nerve in canine acrylamide neuropathy.
Dogs exposed to acrylamide develop a sensorimotor peripheral neuropathy and megaoesophagus. The presence of neuropathy was confirmed electrophysiologically and histologically. Hindlimb motor conduction velocity was reduced and there was a loss of large diameter myelinated fibres in the dorsal common digital nerve and the tibial nerve. The conduction velocity
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23. Nerve fibre velocity and refractory period distributions in nerve trunks.
With the use of a double stimulus technique, nerve fibre velocity range measurements were performed over a single conduction distance in 13 normal subjects and over two conduction distances in another 12 normal subjects. The velocity ranges were found to be dependent upon the conduction distance, owing to unknown refractory period delays. Refractory period v
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24. Effects of lead exposure on peripheral nerve in the cynomolgus monkey.
The relationship between blood lead concentration and nerve conduction velocity has been examined, using the cynomolgus monkey as a model for human lead poisoning, with lead dose and blood lead concentration maintained under controlled conditions, to determine whether nerve conduction velocity could be used as an objective measure of the effects of lead on t