Ketosis
Mostrando 13-24 de 38 artigos, teses e dissertações.
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13. THE ROLE OF THE ENDOCRINE GLANDS IN KETOSIS. II. KETONEMIA FOLLOWING INSULIN HYPOGLYCEMIA*
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14. A−β− Subtype of Ketosis-Prone Diabetes Is Not Predominantly a Monogenic Diabetic Syndrome
American Diabetes Association.
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15. Post-exercise ketosis in post-prandial exercise: effect of glucose and alanine ingestion in humans.
This study examined ketosis in response to 90 min of running before and after the ingestion of 50 g glucose or 50 g L-alanine in thirty-three athletes. Everyone ran 20 km at 07.30 h and then rested, while fasting, till 16.00 h. There were four test groups: 'glucose-before', 'glucose-after', 'alanine-before' and 'alanine-after' according to whether glucose or
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16. CHANGES IN LIVER GLYCOGEN STUDIED BY THE NEEDLE ASPIRATION TECHNIC IN PATIENTS WITH DIABETIC KETOSIS. WITH A METHOD FOR THE ESTIMATION OF GLYCOGEN FROM HISTOLOGIC PREPARATIONS 1
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17. STUDIES OF THE ROLE OF THE LIVER IN HUMAN CARBOHYDRATE METABOLISM BY THE VENOUS CATHETER TECHNIC. II. PATIENTS WITH DIABETIC KETOSIS, BEFORE AND AFTER THE ADMINISTRATION OF INSULIN 12
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18. Ketogenic diets and physical performance
Impaired physical performance is a common but not obligate result of a low carbohydrate diet. Lessons from traditional Inuit culture indicate that time for adaptation, optimized sodium and potassium nutriture, and constraint of protein to 15–25 % of daily energy expenditure allow unimpaired endurance performance despite nutritional ketosis.
BioMed Central.
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19. Pulmonary melioidosis in a patient with diabetes mellitus
A 32-year-old male diabetic developed an acute episode of progressive pulmonary melioidosis while employed in Vietnam. Ketosis and undue glycosuria frequently noted in diabetics with other acute infections were not recognized in this patient. The patient's response to combined and long-term antimicrobials was excellent. This approach to therapy is recommende
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20. Neutrophil Chemiluminescence during Phagocytosis Is Inhibited by Abnormally Elevated Levels of Acetoacetate: Implications for Diabetic Susceptibility to Infections
Human neutrophils by a chemiluminescence assay exhibit diminished phagocytic activity in the presence of abnormally high levels of the serum metabolite acetoacetate. These findings, along with our previous evidence demonstrating myeloperoxidase inhibition by acetoacetate, implicate metabolic ketosis in the inhibition of neutrophil microbicidal activity and t
American Society for Microbiology.
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21. The effects of post-exercise glucose and alanine ingestion on plasma carnitine and ketosis in humans.
1. Several studies have hypothesized that alanine decreases plasma ketone body levels by increasing availability of oxaloacetate, thus allowing acetyl groups to enter the tricarboxylic acid cycle and releasing co-enzyme A (CoA). 2. Four, fasted adult males exercised at 50% of their maximal oxygen consumption for 1.5 h, then ingested 100 g of either glucose o
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22. Acute Reversal of Experimental Diabetic Ketoacidosis in the Rat with (+)-Decanoylcarnitine
The effect of (+)-decanoylcarnitine, a potent inhibitor of long-chain acylcarnitine transferase, was tested for its ability to inhibit hepatic ketogenesis both in the isolated perfused liver and in vivo in severely ketotic alloxan diabetic rats. In vitro the inhibitor caused an almost complete block in ketone body production. In vivo (+)-decanoylcarnitine ca
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23. Beta-adrenergic blockade restores glucose's antiketogenic activity after exercise in carbohydrate-depleted athletes.
1. The development of post-exercise ketosis is not abolished by the ingestion of glucose immediately after exercise, despite inducing high insulin/glucagon ratios in the peripheral (and therefore by implication in the portal) blood. 2. To investigate the possibility of autonomic control of the liver influencing its sensitivity to the major counter-regulatory
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24. Myopathy with abnormal mitochondria, transient low electron transport capacity in the respiratory chain, and absence of energy transduction at sites 1 and 2 in vitro.
A male adult with exercise-related myalgia and weakness from the age of 17 years, developed contractions after moderate exertion which were electrically silent. Triglyceride loading or prolonged fasting provoked excessive ketosis. His isolated muscle mitochondria had severe blockade of the respiratory chain, particularly of NADH-CoQ reductase. After 1.5 year