Impaired Glucose Disturbance
Mostrando 1-5 de 5 artigos, teses e dissertações.
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1. Índice glicêmico da dieta habitual e alteração da homeostase glicêmica em nipo-brasileiros de Bauru / Dietary glycemic index in relation to impaired glucose homeostasis disturbances in Japanese-Brazilians living in Bauru.
Objetivos. Investigar a associação entre consumo alimentar, índice glicêmico (IG) da dieta habitual com glicemia e insulinemia de jejum, resistência à insulina (HOMA R) e alteração da homeostase glicêmica (AHG: glicemia de jejum alterada - GJA, tolerância à glicose diminuída - TGD e diabetes mellitus tipo 2). Indivíduos e métodos. 1.054 nipo-br
Publicado em: 2005
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2. Observations on cardiovascular and neuroendocrine disturbance in the Guillain-Barré syndrome
Cardiovascular disturbances were found to be a common feature of patients with the Guillian-Barré syndrome who were severely paralysed, requiring assisted ventilation. Glycosuria was noted in association with these disturbances, and in five patients investigated we found impaired glucose tolerance tests at the height of the paralysis. Catecholamine and 17-h
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3. Reversal of beta-cell suppression in vitro in pancreatic islets isolated from nonobese diabetic mice during the phase preceding insulin-dependent diabetes mellitus.
Insulin-dependent diabetes mellitus (IDDM) is characterized by a progressive autoimmune destruction of the pancreatic beta-cells. One of the best-suited animal models for IDDM is the nonobese diabetic (NOD) mouse. In this investigation pancreatic islets were isolated from female NOD mice aged 5-7, 8-11, and 12-13 wk and examined immediately (day 0) or after
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4. Tertiary hypothyroidism and hyperglycemia in mice with targeted disruption of the thyrotropin-releasing hormone gene
Thyrotropin-releasing hormone (TRH) is a brain hypothalamic hormone that regulates thyrotropin (TSH) secretion from the anterior pituitary and is ubiquitously distributed throughout the brain and other tissues including pancreas. To facilitate studies into the role of endogenous TRH, we have used homologous recombination to generate mice that lack TRH. These
The National Academy of Sciences of the USA.
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5. Adult-onset degeneration of adipose tissue in mice deficient for the Sox8 transcription factor
Although the transcription factor Sox8 is broadly expressed during embryogenesis in developing ectodermal and mesodermal tissues, mice develop surprisingly normally in the absence of Sox8. Phenotypes in adult Sox8-deficient mice include mild osteopenia, late-onset male infertility, and reduced weight. We show here that progressive degeneration of adipose tis
American Society for Biochemistry and Molecular Biology.