Immunodepression
Mostrando 13-24 de 41 artigos, teses e dissertações.
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13. Immunodepression in the surgical patient and increased susceptibility to infection
Multiple organ failure is the major problem in intensive care patients. The failure of the organ 'immune system' is frequently overlooked, however. In this issue the article by Angele and Faist provides an excellent review of the topic. Deactivation of monocyte and lymphocyte functions seems to play a key role in post-traumatic immunodepression. To accompany
BioMed Central.
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14. Catecholamine-induced interleukin-10 release: a key mechanism in systemic immunodepression after brain injury
BioMed Central.
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15. Suppression of primary antibody response to sheep erythrocytes in susceptible and resistant mice infected with Giardia muris.
The kinetics of anti-sheep erythrocyte (SRBC) response were studied in susceptible (A/J) and resistant (B10.A) mice during infection with Giardia muris. Mice infected with G. muris were found to be less responsive to either intraperitoneally or intraduodenally administered SRBC. Immunodepression was of relatively short duration, occurring during the period o
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16. Immunodepression in Taenia crassiceps infection: restoration of the in vitro response to sheep erythrocytes by activated peritoneal cells.
The in vitro response to sheep erythrocytes of mesenteric lymph node cells from mice infected with the larval cestode Taenia crassiceps is significantly depressed and can be restored to control levels by addition of activated peritoneal cells depleted of functional T or B lymphocytes. Adherent mesenteric lymph node cells from infected mice are unable to reco
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17. Clinical review: Immunodepression in the surgical patient and increased susceptibility to infection
Several studies indicate that organ failure is the leading cause of death in surgical patients. An excessive inflammatory response followed by a dramatic paralysis of cell-mediated immunity following major surgery appears to be responsible for the increased susceptibility to subsequent sepsis. In view of this, most of the scientific and medical research has
BioMed Central.
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18. Depressed antibody responses to a thymus-dependent antigen in toxoplasmosis.
The immunodepressive effect of Toxoplasma gondii infection in mice was studied, using sheep erythrocytes (SRBC) as the testing antigen and serum hemagglutinins, hemolysins, and both direct and indirect splenic plaque-forming cells (PFC) to SRBC as assays. In the primary antibody response, immunoglobulin M (IgM), hemagglutinins, and hemolysins and both IgM- a
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19. Immunodepression by Rowson-Parr Virus in Mice. II. Effect of Rowson-Parr Virus Infection on the Antibody Response to Sheep Red Cells In Vivo and In Vitro
The effects of infection with Rowson-Parr virus (RPV), an associated virus present in Friend virus (FV) preparations, on the splenic plaque-forming cell response to sheep red cells in adult mice and on the plaque-forming cell response of peritoneal cells in vitro have been investigated. The results support the hypothesis that RPV might be responsible for a c
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20. Acquired Cell-Mediated Immunodepression in Acute Chagas' Disease
In this study two groups of patients with acute Chagas' disease were identified. Group one consisted of five patients with apparent acute Chagas' disease. These patients showed symptoms and signals of an acute illness, such as high fever and enlarged spleen. One of these patients developed severe myocarditis and heart failure. Group two consisted of seven pa
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21. Immunodepression by Rowson-Parr Virus in Mice. I. Growth Curves of Rowson-Parr Virus and Immunological Relationships with Friend Virus
The growth curve of Rowson-Parr virus (RPV) was studied in the spleen of mice infected with RPV only and of mice infected with Friend virus (FV) preparations, which consist of a mixture of RPV and FV. In mice infected with FV preparations, RPV replicated to a higher titer than FV and attained higher levels than in singly infected mice. The chronic viremia of
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22. Friend virus-induced immunodepression: effect of neuraminidase treatment on Thy-1.2 antigen expression and cytotoxic potential of splenocytes from virus-infected mice.
Infection of susceptible strains of mice with Friend leukemia virus (FLV) results in a profound depression of cell-mediated immunity as assessed by lymphocyte-mediated cytotoxicity. This depression occurs early in the disease, before the onset of splenomegaly, and is associated with a decline in the susceptibility of splenocytes from FLV-infected mice to lys
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23. Modification of T-cell proliferation and interleukin 2 production in mice infected with Trypanosoma cruzi.
Acute infection of mice with Trypanosoma cruzi results in severe immunodepression and the appearance of autoimmune symptoms. In vitro, concanavalin A-stimulated T cells from spleens of infected animals could neither produce nor respond to interleukin 2. Interleukin 2 production was not restored by addition of exogenous interleukin 1, and proliferative respon
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24. Immunodepression by Rowson-Parr virus in mice; lymphocyte markers and capping response of spleen and lymph node cells after infection.
Infection with Rowson-Parr virus (RPV) induced a rapid reduction in the number of immunoglobulin-positive and theta antigen-positive cells detectable by immunofluorescence in the spleens of susceptible BALB/c mice. The changes produced by RPV infection in the lymph nodes were different, since the number of immunoglobulin-positive cells was increased and the