Fecal Peritonitis
Mostrando 13-24 de 25 artigos, teses e dissertações.
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13. Efeitos do jejum agudo ou jejum intermitente na evolução da peritonite bacteriana induzida por ligadura e punção do ceco ou por injeção intra-peritoneal de suspensão fecal em camundongos / Efeitos do jejum agudo ou jejum intermitente na evolução da peritonite bacteriana induzida por ligadura e punção do ceco ou por injeção intra-peritoneal de suspensão fecal em camundongos
Intermittent fasting is frequent in medical practice and this condition has been studied as a therapeutic intervention for some diseases. Increased life span and resistance to stress is observed in rodents submitted to intermittent fasting. However there is not much information on the evolution of infections in animals submitted to these diet manipulations.
Publicado em: 2007
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14. Estudo comparativo da celularidade da reação inflamatória aguda na serosa e mesentério intestinais às telas de poliamida e polipropileno em ratos wistar com peritonite fecal por técnica morfométrica
Laparostomy constitutes in important strategy in the management of patients with severe acute peritonitis and compartmental abdominal syndrome. Prosthetic materials are used to close the abdominal cavity temporally and the polyamide has been little mention in medical literature. Researches are about chronic inflammatory response. However in laparostomy the p
Publicado em: 2004
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15. Resposta terapÃutica e inflamatÃria de ratos com infecÃÃo peritoneal submetidos ao uso tÃpico de Ampicilina/Sulbactam
A peritonite aguda representa uma importante causa de sepsis e Ãbito nas unidades de terapia intensiva e cirurgia. Classicamente o seu tratamento deve incluir: a administraÃÃo sistÃmica de antibiÃticos, a remoÃÃo mecÃnica dos XXII contaminantes e a restauraÃÃo da integridade gastrintestinal. A utilizaÃÃo de antibiÃticos diretamente na cavidade p
Publicado em: 2004
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16. Colony-stimulating factor 1-dependent resident macrophages play a regulatory role in fighting Escherichia coli fecal peritonitis.
Osteopetrotic op/op mice have less than 5% of the normal number of macrophages in the peritoneal cavity (W. Wiktor-Jedrzejczak, A. Ahmed, C. Szczylik, and R.R. Skelly, J. Exp. Med. 156:1516-1527, 1982). Fecal peritonitis was induced by intraperitoneal injection of 0.5 ml of 5% autoclaved feces in saline along with Escherichia coli grown from feces of mice of
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17. Therapeutic Effects of Nitric Oxide Inhibition during Experimental Fecal Peritonitis: Role of Interleukin-10 and Monocyte Chemoattractant Protein 1
This study demonstrates that the therapeutic effect of a nitric oxide inhibitor in a murine model of fecal peritonitis is mediated in part by increased levels of interleukin-10 (IL-10) and monocyte chemoattractant protein 1 (MCP-1). Female CD1 mice were subjected to cecal ligation and puncture (CLP) with a 21-gauge needle and, immediately following surgery,
American Society for Microbiology.
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18. Microbiological diagnosis of peritonitis in patients on continuous ambulatory peritoneal dialysis.
The fast and accurate etiological diagnosis of peritonitis in patients on continuous ambulatory peritoneal dialysis is essential. The concentration of larger volumes of peritoneal fluids may yield more isolates than conventional methods. The removal of antibiotics present in the fluids as a consequence of therapy by washing or using antibiotic-removing resin
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19. Characterization of Escherichia coli hemolysins conferring quantitative differences in virulence.
Recombinant plasmids encoding hemolysins (hly) isolated from four different Escherichia coli strains were found to be very similar by restriction endonuclease fragment analysis within the hemolysin region. Each of the four recombinant plasmids were used to transform a nonhemolytic fecal strain of E. coli. The comparative virulence of the transformants was te
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20. Contribution of Escherichia coli Alpha-Hemolysin to Bacterial Virulence and to Intraperitoneal Alterations in Peritonitis
Alpha-hemolysin (Hly) is a common exotoxin produced by Escherichia coli that enhances virulence in a number of clinical infections. The addition of hemolysin production to laboratory bacterial strains is known to increase the lethality of E. coli peritonitis. However, the mechanisms involved have not been determined and the contribution of hemolysin to the a
American Society for Microbiology.
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21. Experimental Intra-Abdominal Abscesses in Rats: Quantitative Bacteriology of Infected Animals
An animal model simulating intra-abdominal sepsis was produced by implanting large bowel contents into the pelvic region of rats. Bacteriological analysis of infected sites showed quantitative differences according to the stage of disease. During the initial, often lethal, peritonitis stage, Escherichia coli (mean concentration, 106/ml), enterococci (105) an
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22. Arginine vasopressin in septic shock: supplement or substitute for norepinephrine?
In the current issue of Critical Care, Simon and coworkers investigated the effects of first-line arginine vasopressin (AVP) on organ function and systemic metabolism compared with norepinephrine in a pig model of fecal peritonitis. AVP was titrated according to the mean arterial pressure suggesting a vasopressor rather than a hormone replacement therapy. Th
BioMed Central.
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23. Two related strains of feline infectious peritonitis virus isolated from immunocompromised cats infected with a feline enteric coronavirus.
Two groups of cats were experimentally infected orally with the cat-passaged RM strain of feline enteric coronavirus (FECV-RM). One group of cats (n = 19) had been chronically infected with feline immunodeficiency virus (FIV) for over 6 years, while a second control group (n = 20) consisted of FIV-naive siblings. Fecal virus shedding of FECV occurred in both
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24. Rapid emergence of quinolone resistance in cirrhotic patients treated with norfloxacin to prevent spontaneous bacterial peritonitis.
We carried out quantitative culturing of stools from 31 hospitalized alcoholic patients with cirrhosis and ascites, before treatment with 400 mg of norfloxacin per day, weekly for the first month, and then every 2 weeks thereafter for 15 to 229 days (median, 54 days). Members of the family Enterobacteriaceae virtually disappeared from the stools (< 10(2)/g),