Factor Xiiia
Mostrando 1-12 de 43 artigos, teses e dissertações.
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1. In situ immune response in human dermatophytosis: possible role of Langerhans cells (CD1a+) as a risk factor for dermatophyte infection
ABSTRACT Dermatophytosis is a cutaneous mycosis caused by a plethora of keratinophilic fungi, but Trichophyton rubrum is the most common etiological agent. Despite its high prevalence worldwide, little is known about the host defense mechanisms in this infection, particularly the in situ immune response. Using an immunohistochemistry approach, we investigate
Rev. Inst. Med. trop. S. Paulo. Publicado em: 10/10/2019
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2. Tissue immunostaining for factor XIIIa in dermal dendrocytes of pityriasis alba skin lesions
BACKGROUND: Pityriasis alba affects 1% of the world population and about 9.9% of the children in Brazil. However, its etiology remains uncertain. OBJECTIVE: The objective of the present study was to evaluate the immunoexpression of factor XIIIa in dermal dendrocytes of skin lesions of pityriasis alba. METHOD: Twenty patients with pityriasis alba and 20
An. Bras. Dermatol.. Publicado em: 2014-04
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3. Células dendríticas plasmocitoides, dendrócitos dérmicos fator XIIIa positivos, macrófagos e expressão da forma induzida da óxido nítrico sintase na resposta tecidual cutânea de leishmaniose tegumentar americana / Plasmacytoid dendritic cells, Factor XIIIa-positive dermal dendrocytes, macrophages and inducible nitric oxide synthase expression in American tegumentary leishmaniasis skin lesions
Em todas as formas clínicas da leishmaniose tegumentar americana os macrófagos são as células efetoras mais importantes na destruição do parasita intracelular. As células dendríticas são células apresentadoras de antígeno localizadas nos sítios de inoculação, como pele e mucosa. Os dendrócitos dérmicos Fator XIIIa positivos são células deri
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 10/08/2012
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4. Interstitial and Langerhans' dendritic cells in chronic periodontitis and gingivitis
The aim of the present study was to compare quantitatively the distribution of dendritic cell subpopulations in chronic periodontitis and gingivitis. Fourteen biopsies from patients with chronic periodontitis and fifteen from patients with gingivitis were studied. An immunoperoxidase technique was used to quantify the number of Langerhans' cells (CD1a) and i
Brazilian Oral Research. Publicado em: 2008-09
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5. Dendritic cells, inducible nitric oxide synthase and citokines expression in pityriasis lichenoides skin lesions / Células dendríticas, expressão da forma induzida da óxido nítrico sintase e padrão de citocinas nas lesões de pitiríase liquenóide
BACKGROUND: Pityriasis lichenoides (PL) is a cutaneous disease of unknown etiology which has been regarded as an immunologically mediated reaction. Recently, it was reclassified in the group of Cutaneous Lymphoid T cell Dyscrasia. There are few reports addressing mainly T cell subsets in PL tissue reaction. METHODS: Skin biopsies taken from 34 patients with
Publicado em: 2008
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6. A resposta inflamatória na urticária aguda associada a medicamentos: avaliação imunoistoquímica e imunoeletrônica da unidade microvascular da derme / The inflammatory response in acute drug-induced urticaria: immunohistochemistry and ultrastructure study of dermal microsvascular unit
INTRODUÇÃO: O conhecimento sobre os tipos celulares envolvidos na patogenia da urticária constitui um elemento essencial para a compreensão da fisiopatologia desta doença. Poucos autores têm dado atenção às interações entre mastócitos e dendrócitos da derme na urticária. Os objetivos deste estudo são orientados no sentido de descreverem-se os
Publicado em: 2007
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7. Factor XIIIa positive dermal dendrocytes in American tegumentary leishmaniasis cutaneous tissue response
Revista do Instituto de Medicina Tropical de São Paulo. Publicado em: 2004-08
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8. Ressecção videolaparoscópica de hemangiopericitoma do estômago
In the present case (77 years-old woman), the diagnosis on an extramucosal lesion by endosonography was leiomyoma or schwanoma. Radiological exam of the upper digestive tract with barium and abdominal computed tomography confirmed the site of the lesion at the level of the lesser curvature. The operative technique followed the steps of the laparoscopic parti
Revista do Colégio Brasileiro de Cirurgiões. Publicado em: 2000-06
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9. Primary structure of blood coagulation factor XIIIa (fibrinoligase, transglutaminase) from human placenta.
We have determined the primary structure of human placental factor XIIIa, an enzyme [fibrinoligase, transglutaminase, fibrin-stabilizing factor, EC 2.3.2.13 (protein-glutamine:amine gamma-glutamyltransferase)] that forms intermolecular isopeptide bonds between fibrin molecules as the last step in blood coagulation. Placental factor XIIIa is an unglycosylated
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10. Characterization of cDNA coding for human factor XIIIa.
A cDNA library prepared from human placenta has been screened for sequences coding for factor XIIIa, the enzymatically active subunit of the factor XIII complex that stabilizes blood clots through crosslinking of fibrin molecules. Two oligonucleotides, based on the amino acid sequences of tryptic peptides of factor XIIIa, were used as hybridization probes. O
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11. Labeling of epsilon-lysine crosslinking sites in proteins with peptide substrates of factor XIIIa and transglutaminase.
Peptides patterned on the N-terminal sequence of fibronectin were synthesized and tested for amine acceptor qualities in reactions with dansylcadaverine catalyzed either by coagulation factor XIIIa or intracellular transglutaminase (protein-glutamine:amine gamma-glutamyltransferase, EC 2.3.2.13). On the basis of inverse half-saturations of the enzymes, the o
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12. Cross-linking of fibronectin to collagen by blood coagulation Factor XIIIa.
Soluble fibronectin is found in body fluids and media of adherent cultured cells and binds to fibrin and collagen. Insoluble fibronectin is found in tissue stroma and in extracellular matrices of cultured cells. Fibronectin is a substrate for Factor XIIIa (plasma transglutaminase) and can be cross-linked by Factor XIIIa to itself and the the alpha-chain of f