Collagen Immunology
Mostrando 1-4 de 4 artigos, teses e dissertações.
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1. Aerobic conditioning inhibits ovalbumin allergic sensitization and airway inflammation in mice / A melhora do condicionamento físico aeróbico na sensibilização e na inflamação e remodelamento das vias aéreas de camundongos
A prevalência de doenças respiratórias alérgicas tem aumentado, embora de origem multifatorial, a atividade física parece representar um importante contribuinte. Objetivo: Nós hipotetizamos que o condicionamento aeróbio (AC) prévio à sensibilização com OVA, poderia reduzir a inflamação das vias aéreas, através de um desbalanço via Th1/Th2 ou
Publicado em: 2009
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2. Morphometric study of immune cellular and pre-acinar axis remodeling by type V collagen induction on bronchiolite obliterans model / Estudo morfométrico da imunidade celular e remodelamento no eixo pré-acinar na indução do colágeno tipo V em um modelo de bronquiolite obliterante
A minority of patients with remodeling process of lungs following bronquiolite obliterante (BO) responds to corticosteroids. So, we sought to validate the importance of type V collagen (tVc) tolerance from immunization as option in BO model treatment. Througt of morphometric analyses, we have mensured for the dimensions, the collagen and cell infiltration de
Publicado em: 2007
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3. Humoral immune response characterization of the type V collagen induced scleroderma experimental model / Caracterização da resposta imune em modelo experimental de esclerodermia induzida por colágeno tipo V
Experimental models for human diseases are of utmost importance, since they allow the study of their pathogenesis and therapeutic approach. In this group we can include the experimental model of collagen V-induced scleroderma, in rabbits, with histological alterations (skin, lung and kidney) similar to those observed in humans. Auto-immune diseases have an u
Publicado em: 2005
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4. Macrophage functions are regulated by murine decidual and tumor extracellular matrices.
Because of their paternal antigens, the fetus and placenta may be considered an allograft in the maternal host. Understanding the mechanisms which prevent maternal immunological rejection of the fetus remains a fundamental unsolved problem in immunology. We have previously reported that macrophages are inhibited by maternal decidual stromal cells residing at