Chronic Pulmonary Thromboembolism
Mostrando 1-12 de 12 artigos, teses e dissertações.
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1. Use of direct oral anticoagulants for chronic thromboembolic pulmonary hypertension
OBJECTIVES: Chronic thromboembolic pulmonary hypertension is one of the most prevalent forms of pulmonary hypertension and is a major complication of acute pulmonary embolism. One mainstay of chronic thromboembolic pulmonary hypertension treatment is lifelong anticoagulation. The recent advent of direct oral anticoagulants for acute pulmonary embolism treat
Clinics. Publicado em: 17/05/2018
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2. O desafio de diagnosticar tromboembolia pulmonar aguda em pacientes com doenças pulmonar obstrutiva crônica / The challenge of diagnosing acute pulmonary thromboembolism in patients with chronic obstructive pulmonary disease
Tromboembolia pulmonar aguda e exacerbação aguda de doença pulmonar obstrutiva crônica são doenças comuns. A doença pulmonar obstrutiva crônica é um fator clínico de risco para tromboembolia pulmonar aguda. As apresentações clínicas da tromboembolia pulmonar aguda e da exacerbação aguda da doença pulmonar obstrutiva crônica freqüentemente m
Publicado em: 2010
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3. Avaliação de tromboembolismo em pós-operatório da cirurgia do labirinto (Cox-Maze) para fibrilação atrial crônica secundária a lesão mitral / Assessment of thromboembolism after the cox-maze prcedure for chronic atrial fibrillation secondary to mitral valve lesion
Objective – To assess the occurrence of late thromboembolism after surgical repair of chronic atrial fibrillation (AF) simultaneously with repair of mitral valve using the Cox-Maze procedure. Methods – 69 patients underwent Cox 3 procedure, with no cryoablation simultaneously with mitral valvuloplasty or prosthesis. Mean age was 49.9±13.2 years. Mean fo
Publicado em: 2010
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4. Chagas heart disease: pathophysiologic mechanisms, prognostic factors and risk stratification
Chagas heart disease (CHD) results from infection with the protozoan parasite Trypanosoma cruzi and is the leading cause of infectious myocarditis worldwide. It poses a substantial public health burden due to high morbidity and mortality. CHD is also the most serious and frequent manifestation of chronic Chagas disease and appears in 20-40% of infected indiv
Memórias do Instituto Oswaldo Cruz. Publicado em: 2009-07
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5. Reatividade da artéria pulmonar, ao óxido nítrico inalado, antes e após a tromboendarterectomia / Pulmonary artery vascular reactivity, after nitric oxide inhalation, before and after pulmonary thromboendarterectomy
INTRODUCTION: After pulmonary embolism, 1 to 3% of these patients develop a chronic embolism, with elevated pulmonary artery pressure and increased pulmonary vascular resistance (PVR). The continuous elevated pressure leads to the remodeling of the small arteries. The objective of this study is to determinate if there is an increase in the responsiveness to
Publicado em: 2009
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6. Assessment of Thromboembolism After the Cox-Maze Procedure for Chronic Atrial Fibrillation Secondary to Mitral Valve Lesion
OBJECTIVE: To assess the occurrence of late thromboembolism after surgical repair of chronic atrial fibrillation (AF) simultaneously with repair of mitral valve using the Cox-Maze procedure. METHODS: 69 patients underwent Cox 3 procedure, with no cryoablation simultaneously with mitral valvuloplasty or prosthesis. Mean age was 49.9±13.2 years. Mean follow-u
Arquivos Brasileiros de Cardiologia. Publicado em: 2002-04
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7. Clinical course and late prognosis of treated subacute massive, acute minor, and chronic pulmonary thromboembolism.
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8. Indications for Thrombolytic Therapy in Acute Pulmonary Embolism
Pulmonary thromboembolism is commonly misdiagnosed and is associated with significant morbidity and mortality both in the early and late stages. A major cause of late morbidity is chronic pulmonary hypertension. Although the incidence of chronic thromboembolic pulmonary hypertension is unknown, there is anatomic and physiologic evidence that it is responsibl
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9. High prevalence of dysfibrinogenemia among patients with chronic thromboembolic pulmonary hypertension
The mechanism by which chronic thromboembolic pulmonary hypertension (CTEPH) develops after acute pulmonary thromboembolism is unknown. We previously reported that fibrin from CTEPH patients is relatively resistant to fibrinolysis in vitro. In the present study, we performed proteomic, genomic, and functional studies on fibrin(ogen) to investigate whether ab
American Society of Hematology.
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10. Arachnoid nodules in the lungs of high altitude Indians.
BACKGROUND--Nodules of cells showing a striking histological similarity to those of arachnoid villi have previously been found closely adjacent to pulmonary venules in several diseases associated with alveolar hypoxia or pulmonary oedema including mitral stenosis, plexogenic pulmonary arteriopathy, pulmonary thromboembolism, and chronic obstructive pulmonary
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11. Perfusion Lung Scans Provide a Guide to Which Patients With Apparent Primary Pulmonary Hypertension Merit Angiography
There is hesitancy, based on the perceived risk, to do pulmonary angiography in patients believed to have primary pulmonary hypertension. Yet pulmonary hypertension due to major-vessel, chronic thromboembolism mimics primary pulmonary hypertension clinically and on standard laboratory tests. Because thromboembolic pulmonary hypertension is potentially remedi
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12. A clinicopathological study of fatal chronic airways obstruction.
A clinicopathological study of 21 patients who died as a result of chronic airways obstruction was carried out. Thirteen patients had been in right ventricular failure for at least one year before death and the other eight patients did not have right ventricular failure. The patients with long-standing right ventricular failure died at a younger age, on aver