Balloon Catheter Injury
Mostrando 1-12 de 23 artigos, teses e dissertações.
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1. Technique of spinal cord compression induced by inflation of epidural balloon catheter in rabbits (Oryctologus cuniculus): efficient and easy to use model
ABSTRACT The most common cause of spinal cord injury are high impact trauma, which often result in some motor impairment, sensory or autonomic a greater or lesser extent in the distal areas the level of trauma. In terms of survival and complications due to sequelae, veterinary patients have a poor prognosis unfavorable. Therefore justified the study of exper
An. Acad. Bras. Ciênc.. Publicado em: 15/08/2016
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2. Alterations in the Renin-Angiotensin System in the contralateral carotid artery after balloon catheter injury / Alterações do Sistema Renina-Angiotensina na artéria carótida contralateral à lesão por cateter balão
Fifteen days after balloon catheter injury Emax of Ang II was increased in the contralateral artery as compared to values obtained in arteries from intact animals. This increase in Ang II Emax was not modified in the absence of the endothelium. The morphological and morphometric characteristics in the contralateral artery were not altered as compared to cont
Publicado em: 2010
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3. Consequências dos tratamentos realizados com celecoxibe, -tocoferol ou losartan sobre a reatividade em carótidas de ratos submetidos à lesão com cateter balão / Consequences of the treatments performed with celecoxib, -tocopherol or losartan on the carotid of reactivity in rats subjected to injury with balloon catheter.
The injury by balloon catheter is a procedure commonly used to study the mechanisms of restenosis. The mechanical stress produced by the passage of the balloon promotes changes both in the injured artery in the contralateral artery. There was an increased density of neurons containing neuropeptides such as substance P (SP) and the Gene Related Peptide of Cal
Publicado em: 2009
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4. Effect of saline infusion for the maintenance of blood volume on pulmonary gas exchange during temporary abdominal aortic occlusion
We analyzed the effects of saline infusion for the maintenance of blood volume on pulmonary gas exchange in ischemia-reperfusion syndrome during temporary abdominal aortic occlusion in dogs. We studied 20 adult mongrel dogs weighing 12 to 23 kg divided into two groups: ischemia-reperfusion group (IRG, N = 10) and IRG submitted to saline infusion for the main
Brazilian Journal of Medical and Biological Research. Publicado em: 07/02/2007
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5. Efeito da Hipertensão Intracraniana sobre a complacência Gástrica de ratos Anestesiados: Cauterização do Fenômeno e dos Mecanismos Neurais / Effect of the intracranial hypertension on gastric compliance of anaesthetized rats: characterization of the phenomenology and neural mechanisms
In humans, intracranial hypertension (ICH) disturbs cardiovascular function and also modifies gastrointestinal physiology as clinically manifested by nausea and vomiting symptoms. Since gastric compliance drives the gastric emptying of liquid which is inhibited by ICH, it was studied the ICH effect on gastric compliance behavior in anesthetized rats and the
Publicado em: 2004
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6. Role of platelets in smooth muscle cell proliferation and migration after vascular injury in rat carotid artery.
Intimal lesion formation was investigated in rats made thrombocytopenic by a single i.p. injection of a polyclonal antibody made against rat platelets that reduced circulating platelet counts to less than 1% of normal. The carotid artery was then denuded of endothelium with a 2 French balloon catheter, after which no platelets were found adhering to the expo
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7. Antioxidant treatment inhibits the development of intimal thickening after balloon injury of the aorta in hypercholesterolemic rabbits.
The effect of the antioxidant butylated hydroxytoluene (BHT) on the accumulation of intimal smooth muscle cells (SMC) and development of intimal thickening after balloon catheter injury of the aorta were studied in rabbits with dietary-induced hyperlipidemia. Two sets of New Zealand White rabbits (eight rabbits in each group) were fed either 0.25% cholestero
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8. Inhibition of smooth muscle cell proliferation in injured rat arteries. Interaction of heparin with basic fibroblast growth factor.
Heparin inhibits smooth muscle cell (SMC) proliferation after arterial injury by mechanisms that have yet to be defined. Since the initiation of SMC proliferation is mediated by basic fibroblast growth factor (bFGF), we have investigated the possibility that heparin inhibits SMC proliferation by displacing bFGF from the arterial wall. Using a rat carotid art
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9. Recovery of prostacyclin production by de-endothelialized rabbit aorta. Critical role of neointimal smooth muscle cells.
Prostacyclin (PGI2) synthetic capacity was assayed at the surface of aortas at various intervals after removal of endothelium with a balloon catheter. Results were correlated with morphologic changes in the vessel wall seen by light microscopy, scanning and transmission electron microscopy. To assay PGI2 synthetic capacity, we applied an incubation chamber t
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10. Induction of angiotensin converting enzyme in the neointima after vascular injury. Possible role in restenosis.
Angiotensin II (Ang II) promotes growth of vascular smooth muscle cells in vitro. Consistent with this, Ang II enhances neointimal proliferation in vivo after vascular injury, while angiotensin converting enzyme (ACE) inhibitors attenuate this process. Since tissue ACE plays a key role in the control of local Ang II production, we examined whether vascular i
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11. Proliferation of smooth muscle cells after vascular injury is inhibited by an antibody against basic fibroblast growth factor.
Proliferation of smooth muscle cells (SMCs) represents an important event in vascular lesion formation. Despite the common belief that growth factors contribute to the development of the atherosclerotic plaque, until now there has been no direct evidence for a role of mitogens in the development of arterial lesions. Balloon catheter injury of the rat carotid
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12. Role of the p21 cyclin-dependent kinase inhibitor in limiting intimal cell proliferation in response to arterial injury.
Arterial injury induces a series of proliferative, vasoactive, and inflammatory responses that lead to vascular proliferative diseases, including atherosclerosis and restenosis. Although several factors have been defined which stimulate this process in vivo, the role of specific cellular gene products in limiting this response is not well understood. The p21