Changes in the human visual evoked potential caused by the anticholinergic agent hyoscine hydrobromide: comparison with results in Alzheimer's disease.
AUTOR(ES)
Bajalan, A A
RESUMO
The anticholinergic drug hyoscine hydrobromide produced a slowing of the P2 and N3 components of the flash visual evoked potential (VEP) in young normals but did not affect the pattern reversal VEP. These results, which are similar to those found in Alzheimer's disease, could be explained by an underlying defect of the cholinergic system.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1028683Documentos Relacionados
- Visual evoked potentials and neopterin: biopterin ratios in urine show a high correlation in Alzheimer's disease.
- A comparison of changes in the nucleus basalis and locus caeruleus in Alzheimer's disease.
- Identical twins with Alzheimer's disease.
- The olfactory bulbs in Alzheimer's disease.
- Down's syndrome and the links with Alzheimer's disease.