Abnormal Eating Behavior
Mostrando 1-5 de 5 artigos, teses e dissertações.
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1. The validity and 4-year test-retest reliability of the Brazilian version of the eating attitudes test-26
In a cross-sectional study conducted four years ago to assess the validity of the Brazilian version of the Eating Attitudes Test-26 (EAT- 26) for the identification of abnormal eating behaviors in a population of young females in Southern Brazil, 56 women presented abnormal eating behavior as indicated by the EAT-26 and the Edinburgh Bulimic Investigation Te
Publicado em: 2010
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2. The validity and 4-year test-retest reliability of the Brazilian version of the Eating Attitudes Test-26
In a cross-sectional study conducted four years ago to assess the validity of the Brazilian version of the Eating Attitudes Test-26 (EAT-26) for the identification of abnormal eating behaviors in a population of young females in Southern Brazil, 56 women presented abnormal eating behavior as indicated by the EAT-26 and the Edinburgh Bulimic Investigation Tes
Brazilian Journal of Medical and Biological Research. Publicado em: 2005-11
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3. Abnormal eating behavior and inappropriate practices for weight control amongst female adolescents in fortaleza / Comportamento alimentar anormal e práticas inadequadas para controle de peso entre adolescentes do sexo feminino de Fortaleza
Objectives: characterize eating habits and possible risk factors associated with Eating Disorders, amongst female adolescents in Fortaleza-CE, Brazil. Methodology: transversal study, with 652 women between 14 and 20 years of age, students of the second year of Middle-level education. The Bulimic Investigatory Test Edinburgh (BITE), the Body Shape Questionnai
Publicado em: 2002
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4. Loss of Gq/11 Family G Proteins in the Nervous System Causes Pituitary Somatotroph Hypoplasia and Dwarfism in Mice
Heterotrimeric G proteins of the Gq/11 family transduce signals from a variety of neurotransmitter and hormone receptors and have therefore been implicated in various functions of the nervous system. Using the Cre/loxP system, we generated mice which lack the genes coding for the α subunits of the two main members of the Gq/11 family, gnaq and gna11, select
American Society for Microbiology.
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5. Fuel-mediated teratogenesis. Use of D-mannose to modify organogenesis in the rat embryo in vivo.
The unique embryotoxic properties of D-mannose have been used as the basis for a new technique to secure precise temporal correlations between metabolic perturbations during organogenesis and subsequent dysmorphogenesis. Conscious, pregnant rats were infused with D-mannose or equimolar amounts of D-glucose by "square wave" delivery during the interval in whi