Degenerative Myopathy
Mostrando 1-8 de 8 artigos, teses e dissertações.
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1. Spontaneous and experimental poisoning by salinomycin in rabbits in Rio de Janeiro State. / Intoxicações natural e experimental por salinomicina em coelhos no Estado do Rio de Janeiro.
An outbreak of salinomycin poisoning in rabbits is described. From a total of 2000 rabbits, at least 27 died after coccidiostatic sulfaquinoxalin was substituted by salinomycin treatment. In the ration fed to the rabbits 26.9 ppm of salinomycin were detected. Clinical signs included anorexia, apathy and slowness with evolution to incoordination and recumbenc
Publicado em: 2008
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2. Intoxicação por monensina em búfalos. / Monensin toxicosis in water buffaloes
O primeiro artigo desse estudo apresenta a ocorrência de um surto de intoxicação por monensina em búfalos de um rebanho misto com bovinos, os quais não foram afetados. Tal fato sugeriu a possibilidade de que búfalos fossem menos tolerantes à monensina que bovinos. Embora com número reduzido de animais experimentais, dados preliminares foram compatív
Publicado em: 2007
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3. Severe diaphragmatic necrosis in 4 horses with degenerative myopathy
Severe diaphragmatic necrosis occurred in horses with degenerative myopathy due to polysaccharide storage myopathy (n = 2), nutritional myopathy (n = 1), and vasculitis (n = 1). Blood gas analysis performed in 1 horse indicated development of respiratory acidosis. Respiratory muscle necrosis can be severe in horses with degenerative myopathy and can lead to
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4. Familial centronuclear myopathy
The clinical and histological features of two Negro brothers with a centronuclear myopathy are described. They bring to 19 the number of cases now reported with this constellation of physical signs and pathological changes in the muscles. A review of these patients suggests the existence of several different diseases causing this picture, though presumably t
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5. Electromyography in nutritional osteomalacic myopathy.
Electromyographic studies in 15 women with nutritional osteomalacia and proximal muscle weakness showed brief duration motor unit action potentials of normal amplitude and increased proportion of polyphasic motor unit potentials in the majority of them. By employing quantitative methods of electromyography, more positive results were obtained, thus reducing
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6. A mutation in the alpha 3 chain of type IX collagen causes autosomal dominant multiple epiphyseal dysplasia with mild myopathy
Multiple epiphyseal dysplasia (MED) is a degenerative cartilage condition shown in some cases to be caused by mutations in genes encoding cartilage oligomeric matrix protein or type IX collagen. We studied a family with autosomal dominant MED affecting predominantly the knee joints and a mild proximal myopathy. Genetic linkage to the COL9A3 locus on chromoso
The National Academy of Sciences.
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7. Genetic Manipulation of Dysferlin Expression in Skeletal Muscle : Novel Insights into Muscular Dystrophy
Mutations in the gene DYSF, which codes for the protein dysferlin, underlie Miyoshi myopathy and limb-girdle muscular dystrophy 2B in humans and produce a slowly progressing skeletal muscle degenerative disease in mice. Dysferlin is a Ca2+-sensing, regulatory protein that is involved in membrane repair after injury. To assess the function of dysferlin in hea
American Society for Investigative Pathology.
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8. The development of mitochondrial medicine.
Primary defects in mitochondrial function are implicated in over 100 diseases, and the list continues to grow. Yet the first mitochondrial defect--a myopathy--was demonstrated only 35 years ago. The field's dramatic expansion reflects growth of knowledge in three areas: (i) characterization of mitochondrial structure and function, (ii) elucidation of the ste